Author

Maisha Adiba

Leibniz Institute of Virology (LIV)ORCID 0000-0002-3473-8981Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 1 Immunology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 3 Organism:human· 3 Approach:experimental assay· 2 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Approach:diffusion model· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

Martin V. Hamann· 2 shared Sabrina M. LeddyiD· 2 shared Ulrike C. LangeiD· 2 shared Adam T. GrundhoffiD· 1 shared Angelique HölzemeriD· 1 shared Cédric FeschotteiD· 1 shared Christoph Schwarz· 1 shared Daniel J. Merk· 1 shared

Activity by Year

2025-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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2 shared papersMicrobiology

Martin V. Hamann

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

HIV-1 promotes cell-to-cell interactions enabling spread from CD4+ T cells to microglia

Friederike Reinsberg, Katharina Schiering, Malte G Lingstaedt, Leonore Mensching, Maisha Adiba, Tom V Kraus, Jan Broder Engler, Imke Liebold, Lidia Bosurgi, Sebastian Schloer, Marcus A. Altfeld, Manuel Alexander Friese, Susanne Krasemann, Ulrike C. Lange, Wilfredo F. García-Beltrán, Angelique Hölzemer

This study shows HIV-1 enhances cell-to-cell transmission from infected CD4+ T cells to microglia by altering surface signals and increasing phagocytosis.

bioRxiv · May 11, 2026 · PreprintImmunology

Selective persistence of HIV-1-infected T cell clones can occur through immune reprogramming driven by defective, transcriptionally active proviruses

Martin V. Hamann, Lisa Brauckmann, Christoph Schwarz, Michael Spohn, Ramon Stoeck, Sabrina M. Leddy, Julie Frouard, Maisha Adiba, Riekje Winzer, Sanamjeet Virdi, Adam T. Grundhoff, Cédric Feschotte, Frauke Muecksch, Nadia R. Roan, Eva Tolosa, U. Schiefer Lange

This study shows that defective HIV-1 proviruses can reprogram CD4+ T cells by altering BACH2 and STAT5B gene expression, leading to immune evasion and persistence in ART-treated individuals.

bioRxiv · Sep 22, 2025 · PreprintMicrobiology

Expression of LTR and LINE1 transposable elements defines atypical teratoid/rhabdoid tumor subtypes

Martin V. Hamann, Shweta Godbole, Maisha Adiba, Sabrina M. Leddy, Jelena Navolić, Ghazaleh Tabatabai, Daniel J. Merk, Ulrike C. Lange, Julia E. Neumann

This study identifies distinct TE transcription profiles in ATRT subtypes, revealing unique patterns in ATRT-MYC that may inform new therapeutic strategies.

Acta Neuropathologica Communications · Jul 22, 2025Peer-reviewedCancer Biology