Author

G. A. Marsh

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 3 Evolutionary Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:host-pathogen & immune response· 4 Approach:experimental assay· 3 Research Focus:cell signaling & trafficking· 2 Assay:proteomics· 2 Organism:bacteria (other)· 1 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1

Frequent Co-Authors

G. W. Moseley· 2 shared L.-F. Wang· 2 shared M. Tachedjian· 2 shared A. Johnson· 1 shared A. M. Rozario· 1 shared A. McAuley· 1 shared A. Nicolas· 1 shared A. R. Harrison· 1 shared

Activity by Year

2017-2020 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 18 mutual co-author links

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2 shared papersMicrobiology

G. W. Moseley

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Antagonism of STAT3 signalling by Ebola virus

A. R. Harrison, M. Dearnley, S. Todd, D. Green, G. A. Marsh, G. W. Moseley

This study identifies how Ebola virus inhibits STAT3 signaling via VP24, using two distinct mechanisms for STAT3 heterodimers and homodimers.

bioRxiv · Aug 10, 2020Microbiology

Unique Evolution of Antiviral Tetherin in Bats

J. A. Hayward, M. Tachedjian, A. Johnson, A. T. Irving, T. B. Gordon, J. Cui, A. Nicolas, I. Smith, V. Boyd, G. A. Marsh, M. L. Baker, L.-F. Wang, G. Tachedjian

This study characterizes the evolution of tetherin in bats, identifying unique structural variants and splice isoforms with antiviral functions.

bioRxiv · Apr 8, 2020Evolutionary Biology

Filoviruses Can Efficiently Infect Human Neuron-Like Cells Without Genetic Adaptation

A. McAuley, M. Tachedjian, G. A. Marsh

This study shows filoviruses can infect human neuron-like cells without genetic changes, with Marburg virus persisting longer, suggesting potential for long-term neurological effects.

bioRxiv · Dec 12, 2019 · PreprintMicrobiology

Viral hijacking of the nucleolar DNA-damage response machinery: a novel mechanism to regulate host cell biology

S. M. Rawlinson, T. Zhao, A. M. Rozario, C. L. Rootes, P. J. McMillan, A. W. Purcell, A. Woon, G. A. Marsh, K. G. Lieu, L.-F. Wang, H. J. Netter, T. D. Bell, C. R. Stewart, G. W. Moseley

The paper identifies a novel viral mechanism that hijacks the nucleolar DNA-damage response pathway by interacting with Treacle to inhibit rRNA biogenesis.

bioRxiv · Nov 17, 2017Microbiology