Author

G. Dias do Vale

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:human· 2 Assay:proteomics· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Organism:Drosophila· 1

Frequent Co-Authors

J. Mcdonald· 2 shared A. C. Crouch· 1 shared B. Gao· 1 shared B. Paul· 1 shared C. Andreani· 1 shared C. Bartolacci· 1 shared C. Behrens· 1 shared C. Zhang· 1 shared

Activity by Year

2021-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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2 shared papersCell Biology

J. Mcdonald

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Author profile →

3 papers

Recent·Most cited

Paraoxonase-like APMAP maintains endoplasmic reticulum-associated lipid and lipoprotein homeostasis

B. Paul, H. Merta, R. Ugrankar-Banerjee, M. Hensley, S. Tran, G. Dias do Vale, J. G. McDonald, S. A. Farber, W. M. Henne

This paper identifies APMAP as an ER-localized antioxidant that maintains lipid and lipoprotein homeostasis, with loss of APMAP causing ER stress, lipid droplet accumulation, and altered lipoprotein maturation across multiple organisms.

bioRxiv · Jan 26, 2024Cell Biology

CLSTN3B enhances adipocyte lipid droplet structure and function via endoplasmic reticulum contact

C. Zhang, M. Ye, K. Melikov, D. Yang, G. Dias do Vale, J. Mcdonald, K. Eckert, M.-J. Lin, X. Zeng

This study identifies CLSTN3B as a key protein for adipocyte lipid droplet maturation by facilitating ER-LD contacts, enhancing lipid storage and metabolic health.

bioRxiv · Jan 20, 2024 · PreprintCell Biology

Targeting de novo lipogenesis and the Lands cycle induces ferroptosis in KRAS-mutant lung cancer

C. Bartolacci, C. Andreani, G. Dias do Vale, S. Berto, M. Melegari, A. C. Crouch, D. L. Baluya, G. Kemble, K. Hodges, J. Starrett, K. Politi, S. L. Starnes, D. Lorenzini, M. G. Raso, L. Solis Soto, , P. P. Scaglioni

This study identifies FASN as a therapeutic target in KRAS-mutant lung cancer by showing its role in lipid metabolism and ferroptosis induction.

bioRxiv · Mar 18, 2021Cancer Biology