Author

Gracie J. Mead

University of OxfordPrimary ORCID profile (0000-0003-3107-4881 · 1 paper)Unclaimed ORCID profile · follow to get notified of new preprints

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Same name, identified by ORCID

Gracie J. MeadiD1 paper · University of Oxford

Biology Disciplines

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Research Focus:cell signaling & trafficking· 2 Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Assay:flow cytometry· 2 Organism:mouse· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:host-pathogen & immune response· 1 Assay:proteomics· 1

Frequent Co-Authors

Audrey GérardiD· 2 shared Adrian Llewellyn HarrisiD· 1 shared Ana Laura ChiodettiiD· 1 shared Andrew MuiriD· 1 shared Anja SchwenzeriD· 1 shared Arianne C. RichardiD· 1 shared Han CaiiD· 1 shared Iain A. McNeishiD· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersImmunology

Audrey GérardORCID

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2 papers

Recent·Most cited

Transient immune landscape remodelling shapes CD8 T-cell priming during infection

Ana Laura Chiodetti, Han Cai, Andrew Muir, Gracie J. Mead, Mariana Borsa, Arianne C. Richard, Audrey Gérard

The paper shows that transient immune architecture changes during infection protect low-affinity CD8 T-cell priming from Treg inhibition, ensuring broad T-cell responses.

bioRxiv · Mar 20, 2026 · PreprintImmunology

The breast tumor microenvironment exploits eosinophil plasticity to suppress their anti-tumor activity

Zofia Varyova, Mathilde Pohin, Gracie J. Mead, Libby K. Jennings, Valeria da Costa, Sarah Spear, Iain A. McNeish, Anja Schwenzer, Adrian Llewellyn Harris, Audrey Gérard, Kim S. Midwood

This study shows that the breast tumor microenvironment transforms eosinophils into a less active state by reducing Ly6C expression, impairing their anti-tumor function, and that IFN stimulation can partially restore this phenotype.

bioRxiv · Jan 12, 2026 · PreprintImmunology