Author

J. H. Ahn

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Plant Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:development & morphogenesis· 2 Research Focus:metabolism & bioenergetics· 2 Approach:statistical model· 2 Organism:Arabidopsis· 1 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1

Frequent Co-Authors

A. Biju· 1 shared A. Leduc· 1 shared A. Naji· 1 shared A. P. Montalvo· 1 shared A. V. Rozo· 1 shared B. Bilanges· 1 shared B.-S. Park· 1 shared C. Krapp· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 22 mutual co-author links

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1 shared paperCell Biology

A. Biju

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

DEC1 is an adult clock regulator that links circadian rhythms to functional β-cell maturation

Z. Gao, M. Liu, A. P. Montalvo, Z. L. Gruskin, A. Leduc, Y. Xie, S. Preza, J. Du, X. Liu, V. Huhn, Z. Cheng, C. Krapp, M. Haemmerle, A. V. Rozo, X. Wen, , J. R. Alvarez-Dominguez

This study shows DEC1 regulates circadian rhythms in β-cells, linking them to metabolic maturation and glucose responsiveness.

bioRxiv · Aug 11, 2023 · PreprintCell Biology

A merger between compatible but divergent genomes supports allopolyploidization in the Brassicaceae family

H. Shin, J. E. Park, H. R. Park, W. L. Choi, S. H. Yu, W. Koh, S. Kim, H. Y. Soh, N. E. Waminal, H. R. Belandres, J. Y. Lim, G. Yi, J. H. Ahn, J.-S. Kim, Y.-M. Kim, , J. H. Huh

This study investigates how genome divergence and compatibility contribute to the stabilization of hybrid genomes in an allopolyploid plant species.

bioRxiv · Sep 28, 2021 · PreprintPlant Biology

Constitutive activation of the PI3K-Akt-mTORC1 pathway sustains the m.3243A>G mtDNA mutation

C.-Y. Chung, K. Singh, V. N. Kotiadis, G. E. Valdebenito, J. H. Ahn, E. Topley, J. Tan, W. D. Andrews, B. Bilanges, R. D. S. Pitceathly, G. Szabadkai, M. Yuneva, M. R. Duchen

The paper shows that the m.3243A>G mtDNA mutation activates the PI3K-Akt-mTORC1 pathway, which can be inhibited to reduce mutant mtDNA load and improve cellular function.

bioRxiv · Jun 18, 2020 · PreprintCell Biology