Author

K. E. Inouye

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Physiology· 2 Biochemistry· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:cell signaling & trafficking· 2 Research Focus:metabolism & bioenergetics· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:gene regulatory network inference· 1 Approach:genetic perturbation / CRISPR· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

G. S. Hotamisligil· 3 shared A. Lee· 2 shared G. Y. Lee· 2 shared K. J. Prentice· 2 shared A. P. Arruda· 1 shared C. Dominguez-Gonzalez· 1 shared C. Rosique· 1 shared G. Parlakgul· 1 shared

Activity by Year

2022-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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3 shared papersPhysiology

G. S. Hotamisligil

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3 papers

Recent·Most cited

Ubiquinone deficiency drives reverse electron transport to disrupt hepatic metabolic homeostasis in obesity

R. L. S. Goncalves, Z. B. Wang, K. E. Inouye, Y. G. Lee, X. Fu, J. Saksi, C. Rosique, G. Parlakgul, A. P. Arruda, T. S. Hui, M. C. Loperena, S. C. Burgess, I. Graupera, G. S. Hotamisligil

The paper shows that ubiquinone deficiency in obesity leads to reverse electron transport, increasing mROS and disrupting hepatic metabolic homeostasis.

bioRxiv · Feb 21, 2023 · PreprintBiochemistry

ATGL is differentially required for adipocyte FABP4 secretion in vivo and ex vivo

K. J. Prentice, A. Lee, P. Cedillo, K. E. Inouye, M. E. Ertunc, G. Y. Lee, G. S. Hotamisligil

This study shows that ATGL is not required for lipolysis-stimulated FABP4 secretion from adipocytes, which can occur via sympathetic signaling.

bioRxiv · Oct 14, 2022 · PreprintPhysiology

Endothelial FABP4 constitutes the majority of basal circulating hormone levels and regulates lipolysis-driven insulin secretion

K. E. Inouye, K. J. Prentice, A. Lee, C. Dominguez-Gonzalez, M. X. Chen, G. Y. Lee, G. S. Hotamisligil

This study identifies endothelial cells as the main source of baseline circulating FABP4 and shows they are required for lipolysis-driven insulin secretion.

bioRxiv · Oct 13, 2022 · PreprintPhysiology