Author

K. Ghoreschi

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2 Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:human· 3 Research Focus:cell type annotation· 2 Research Focus:host-pathogen & immune response· 2 Assay:proteomics· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell-cell communication· 1

Frequent Co-Authors

A. N. R. Weber· 2 shared F. Herster· 2 shared K. Schaekel· 2 shared M. W. Loeffler· 2 shared Z. Bittner· 2 shared A. M. Sigmund· 1 shared A. S. Yazdi· 1 shared D. Eisel· 1 shared

Activity by Year

2019-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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2 shared papersImmunology

A. N. R. Weber

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Author profile →

3 papers

Recent·Most cited

Apremilast prevents blistering in human epidermis by stabilization of keratinocyte adhesion in pemphigus.

A. M. Sigmund, M. Winkler, S. Engelmayer, D. T. Egu, D. Kugelmann, M. Y. Radeva, F. C. Bayerbach, S. Kotschi, S. Yeruva, E. Schmidt, A. S. Yazdi, K. Ghoreschi, F. Vielmuth, J. Waschke

This study shows apremilast stabilizes keratinocyte adhesion in pemphigus by phosphorylating plakoglobin at serine 665, preventing blistering without affecting desmoglein depletion or binding.

bioRxiv · Feb 7, 2022Cell Biology

Neutrophil-extracellular trap-associated RNA (naRNA) and LL37 enable self-amplifying inflammation in psoriasis

F. Herster, Z. Bittner, S. Dickhoefer, D. Eisel, T. Eigenbrod, T. Knorpp, N. Schneiderhan-Marra, M. W. Loeffler, H. Kalbacher, D. Hartl, L. Freund, K. Schaekel, M. Heiser, K. Ghoreschi, A. N. R. Weber

The paper shows RNA-LL37 complexes activate neutrophils via TLR8, leading to cytokine release and NET formation, which may drive self-sustaining inflammation in psoriasis.

bioRxiv · Feb 19, 2019 · PreprintImmunology

Platelet-neutrophil aggregates promote skin pathology in psoriasis

F. Herster, Z. Bittner, M. C. Codrea, N. Archer, M. Heister, M. W. Loeffler, S. Heumos, J. Wegner, R. Businger, M. Schindler, D. Stegner, K. Schaekel, S. Grabbe, K. Ghoreschi, L. Miller, A. N. R. Weber

This study identifies platelet-neutrophil aggregates as contributors to psoriasis skin pathology and disease severity.

bioRxiv · Jan 28, 2019 · PreprintImmunology