Author

H. Kalbacher

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2 Biochemistry· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:cell signaling & trafficking· 2 Research Focus:host-pathogen & immune response· 2 Organism:human· 2 Assay:proteomics· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1

Frequent Co-Authors

F. Herster· 2 shared M. W. Loeffler· 2 shared S. Dickhoefer· 2 shared A. Delor· 1 shared A. Marcu· 1 shared A. N. R. Weber· 1 shared A. N. Weber· 1 shared A. Tapia Abellan· 1 shared

Activity by Year

2019-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 31 mutual co-author links

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2 shared papersImmunology

F. Herster

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

A network of cytosolic (co)chaperones promotes the biogenesis of mitochondrial signal-anchored outer membrane proteins

D. Rapaport, L. Drwesh, B. Heim, M. Graf, L. Kehr, L. Hansen-Palmus, M. Franz-Wachtel, B. Macek, H. Kalbacher, J. Buchner

This study identifies cytosolic (co)chaperones that interact with signal-anchored mitochondrial proteins and facilitate their biogenesis.

bioRxiv · Feb 15, 2022Biochemistry

BTK operates a phospho-tyrosine switch to regulate NLRP3 inflammasome activity

Z. A. Bittner, X. Liu, S. Shankar, A. Tapia Abellan, H. Kalbacher, L. Andreeva, M. Mangan, P. Duewell, M. Lovotti, K. Bosch, S. Dickhoefer, A. Marcu, S. Stevanovic, F. Herster, M. W. Loeffler, , A. N. Weber

The paper shows BTK modulates NLRP3 inflammasome activity through phosphorylation of tyrosine residues, affecting localization, oligomerization, and IL-1β release.

bioRxiv · Jun 25, 2020Immunology

Neutrophil-extracellular trap-associated RNA (naRNA) and LL37 enable self-amplifying inflammation in psoriasis

F. Herster, Z. Bittner, S. Dickhoefer, D. Eisel, T. Eigenbrod, T. Knorpp, N. Schneiderhan-Marra, M. W. Loeffler, H. Kalbacher, D. Hartl, L. Freund, K. Schaekel, M. Heiser, K. Ghoreschi, A. N. R. Weber

The paper shows RNA-LL37 complexes activate neutrophils via TLR8, leading to cytokine release and NET formation, which may drive self-sustaining inflammation in psoriasis.

bioRxiv · Feb 19, 2019 · PreprintImmunology