Author

Luke Slind

Oregon Health & Science University

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 4

Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Organism:human· 4 Research Focus:host-pathogen & immune response· 3 Assay:proteomics· 1

Frequent Co-Authors

Meaghan H. HancockiD· 4 shared Nicole L. DigginsiD· 4 shared Patrizia CaposioiD· 4 shared Rebekah TurneriD· 4 shared Jennifer MitchelliD· 3 shared Christopher J. Parkins· 2 shared Daniel MalouliiD· 2 shared Daniel N. StreblowiD· 2 shared

Activity by Year

2025-2026 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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4 shared papersMicrobiology

Meaghan H. HancockORCID

Mutual co-authors in cluster

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4 papers

Recent·Most cited

Human cytomegalovirus microRNAs target the Wnt signaling protein Dishevelled 3 to promote efficient reactivation in CD34+ hematopoietic progenitor cells

Nicole L. Diggins, Luke Slind, Jennifer Mitchell, Andrew Pham, Devin N. Fachko, Yan Chen, Rebekah Turner, Patrizia Caposio, Rebecca L. Skalsky, Meaghan H. Hancock

This study shows that HCMV miRNAs miR-US5-2 and miR-US25-2 downregulate Dvl3 during latency to enable efficient reactivation in CD34+ HPCs.

bioRxiv · Oct 19, 2026 · PreprintMicrobiology

The human cytomegalovirus chemokine binding protein UL22A is necessary for efficient reactivation from latency in CD34+ hematopoietic progenitor cells.

Rebekah Turner, Nicole L. Diggins, Luke Slind, Jennifer Mitchell, Andrew H. Pham, Christopher J. Parkins, Wilma D. Perez, Samuel Medica, Michael C. Denton, Takeshi F. Andoh, Gabriela M. Webb, Daniel Andrade-Vera, Daniel N. Streblow, Patrizia Caposio, Meaghan H. Hancock

This paper shows that HCMV's UL22A protein is essential for reactivation from latency in CD34+ hematopoietic progenitor cells, with tyrosine residues critical for this process, but RANTES neutralization does not fully restore reactivation.

Journal of Virology · Jul 27, 2026Peer-reviewedMicrobiology

HCMV promotes viral reactivation through the coordinated regulation of Notch signaling by UL8 and miR-UL36

Aaron T. Dirck, Nicole L. Diggins, Wilma D. Perez, Christopher J. Parkins, Michael Daily, Rebekah Turner, Luke Slind, Linh Nguyen, Daniel Malouli, Guanming Wu, Meaghan H. Hancock, Patrizia Caposio

This study shows HCMV uses UL8 and miR-UL36 to suppress Notch signaling, promoting reactivation in CD34+ hematopoietic progenitor cells.

mBio · Feb 9, 2026Peer-reviewedMicrobiology

Human Cytomegalovirus UL78 is a Nuclear-Localized GPCR Necessary for Efficient Reactivation from Latent Infection in CD34+ Hematopoietic Progenitor Cells

Samuel Medica, Nicole L. Diggins, Michael C. Denton, Rebekah Turner, Lydia J. Pung, Adam T. Mayo, Olivia Kramer Hansen, Jennifer Mitchell, Luke Slind, Linh K. Nguyen, Teresa Beechwood, Gauthami Sulgey, Craig N. Kreklywich, Daniel Malouli, METTE MARIE ROSENKILDE, , Meaghan H. Hancock

This study identifies UL78 as a nuclear-localized GPCR essential for HCMV reactivation from latency in CD34+ HPCs, showing its Gi coupling and interactome involvement in nuclear processes.

Journal of Virology · Oct 8, 2025Peer-reviewedMicrobiology