Author

M. J. Drummond

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

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Physiology· 4

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Approach:experimental assay· 4 Organism:mouse· 4 Research Focus:metabolism & bioenergetics· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Assay:metabolomics· 1 Assay:proteomics· 1

Frequent Co-Authors

H. Eshima· 4 shared J. A. Maschek· 4 shared J. E. Cox· 4 shared K. Funai· 4 shared P. J. Ferrara· 3 shared P. Siripoksup· 3 shared J. L. Shahtout· 2 shared J. M. Johnson· 2 shared

Activity by Year

2019-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 44 mutual co-author links

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4 shared papersPhysiology

H. Eshima

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4 papers

Recent·Most cited

Inhibition of skeletal muscle Lands cycle ameliorates weakness induced by physical inactivity

J. L. Shahtout, H. Eshima, P. J. Ferrara, J. A. Maschek, J. E. Cox, M. J. Drummond, K. Funai

This study shows that inhibiting the Lands cycle in skeletal muscle reduces lipid hydroperoxide levels, protecting against disuse-induced atrophy and weakness in mice.

bioRxiv · Jul 25, 2023Physiology

Phosphatidylethanolamine facilitates mitochondrial pyruvate entry to regulate metabolic flexibility

P. Siripoksup, G. Cao, A. A. A. Cluntun, J. A. Maschek, Q. Pearce, M. J. Lang, H. Eshima, P. C. Opurum, Z. S. Mahmassani, E. B. Taylor, J. E. Cox, M. J. Drummond, J. Rutter, K. Funai

This study identifies phosphatidylethanolamine's role in regulating mitochondrial pyruvate entry, linking it to metabolic flexibility in skeletal muscle.

bioRxiv · Nov 1, 2022 · PreprintPhysiology

Lipid hydroperoxides promote sarcopenia through carbonyl stress

H. Eshima, J. L. Shahtout, P. Siripoksup, M. J. Pearson, Z. S. Mahmassani, P. J. Ferrara, A. W. Lyons, J. A. Maschek, A. D. Peterlin, A. R. P. Verkerke, J. M. Johnson, A. Salcedo, J. J. Petrocelli, E. R. Miranda, E. J. Anderson, , K. Funai

This study shows lipid hydroperoxides contribute to sarcopenia by causing carbonyl stress, and their neutralization prevents muscle atrophy and weakness.

bioRxiv · Dec 17, 2021Physiology

Mitochondrial PE potentiates respiratory enzymes to amplify skeletal muscle aerobic capacity

T. D. Heden, J. M. Johnson, P. J. Ferrara, H. Eshima, A. R. Verkerke, E. J. Wentzler, P. Siripoksup, T. M. Narowski, C. B. Coleman, C.-T. Lin, T. E. Ryan, P. T. Reidy, L. E. de Castro Bras, C. M. Karner, C. F. Burant, , K. Funai

The paper shows that mitochondrial phospholipids, specifically phosphatidylethanolamine (PE), regulate respiratory capacity in skeletal muscle, with PE deficiency causing respiratory failure and oxidative stress.

bioRxiv · Apr 9, 2019Physiology