Author

M. Potempa

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:clustering· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Assay:flow cytometry· 1 Research Focus:host-pathogen & immune response· 1 Organism:mouse· 1

Frequent Co-Authors

A. Rogers· 1 shared C. A. Schiffer· 1 shared C. W. Carter· 1 shared D. Wethington· 1 shared E. A. Nalivaika· 1 shared E. Spielvogel· 1 shared G. Giuliani· 1 shared J. C. Sun· 1 shared

Activity by Year

2018-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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1 shared paperBiochemistry

A. Rogers

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Author profile →

2 papers

Recent·Most cited

Clonal stochasticity in early NK cell response to mouse cytomegalovirus is generated by mature subsets of varying proliferative ability

D. Wethington, S. Ahmad, M. Potempa, G. Giuliani, O. A. Aguilar, M. Poudel, S. Grassmann, W. Stewart, N. M. Adams, J. C. Sun, L. L. Lanier, J. Das

This study quantifies how clonal stochasticity in NK cell responses to MCMV arises from proliferation and death dynamics of mature NK subsets.

bioRxiv · Sep 7, 2023Immunology

HIV-1 Protease Uses Bi-Specific S2/S2’ Subsites To Optimize Cleavage of Two Classes of Target Sites

M. Potempa, S.-K. Lee, N. KurtYilmaz, E. A. Nalivaika, A. Rogers, E. Spielvogel, C. W. Carter, C. A. Schiffer, R. Swanstrom

This study shows HIV-1 protease uses bi-specific S2/S2’ subsites to cleave two classes of target sites, with distinct interactions based on the presence or absence of a P1 proline.

bioRxiv · Aug 29, 2018Biochemistry