Author

M. Quante

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Microbiology· 2 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Organism:human· 3 Research Focus:cell type annotation· 2 Assay:metagenomics· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1

Frequent Co-Authors

K. Steiger· 4 shared R. M. Schmid· 3 shared D. Haller· 2 shared D. Schult· 2 shared K. Neuhaus· 2 shared K.-P. Janssen· 2 shared P. Koyumdzhieva· 2 shared S. Reitmeier· 2 shared

Activity by Year

2021-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 38 mutual co-author links

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Curved chords = mutual co-authorship

4 shared papersMicrobiology

K. Steiger

Mutual co-authors in cluster

Author profile →

5 papers

Recent·Most cited

KRas, in addition to Tp53 is a driver for early carcinogenesis and a molecular target in a mouse model of invasive gastro-esophageal adenocarcinoma

A. Anand, L. Schoemig, S. Lange, L. Tran, K. Flisikowski, R. Oellinger, R. Rad, M. Vieth, K. Steiger, T. Baumeister, J. Strangmann, H. Alguel, K. Ciecielski, K. Boettcher, H.-Y. Fang, , M. Quante

This study identifies KRAS mutation as a driver of invasive gastro-esophageal adenocarcinoma in a mouse model, showing its synergistic role with Tp53 or Rb1 loss and potential as a therapeutic target.

bioRxiv · May 7, 2024 · PreprintCancer Biology

Gut microbial disruption in critically ill patients with COVID-19 associated pulmonary aspergillosis

D. Schult, C. Maurer, P. Koyumdzhieva, S. Reitmeier, M. Middelhoff, S. Rasch, M. List, K.-P. Janssen, K. Steiger, U. Protzer, R. M. Schmid, K. Neuhaus, D. Haller, M. Quante, T. Lahmer

This study investigates the gut and bronchial microbiota in critically ill COVID-19 patients with and without CAPA, finding reduced gut diversity and increased Staphylococcus epidermidis in CAPA patients.

bioRxiv · Oct 8, 2022Microbiology

Intestinal myofibroblasts regulate intestinal epithelial cell plasticity via YAP/TAZ

A. Pastuła, K. P. Janssen, K. Steiger, J. Slotta-Huspenina, S. M. Hauck, R. M. Schmid, M. Quante, M. Borgmann, M. J. A. Ruiz-Fernandez, T. C. Wang, R. A. Friedman, U. Ehmer, M. Fottner

This study shows that intestinal myofibroblasts enhance enteroid self-renewal and survival through YAP/TAZ signaling and TGF-β, without involving Wnt.

bioRxiv · Oct 7, 2022 · PreprintCell Biology

Gut Bacterial Dysbiosis and Instability is Associated with the Onset of Complications and Mortality in COVID-19

D. Schult, S. Reitmeier, P. Koyumdzhieva, M. Middelhof, J. Erber, J. Schneider, J. Krager, M. Frolova, J. Horstmann, L. Fricke, K. Steiger, M. Jesinghaus, K.-P. Janssen, U. Protzer, K. Neuhaus, , M. Quante

The paper analyzes gut and oral microbiota in COVID-19 patients to show associations between microbial dysbiosis, complications, and mortality.

bioRxiv · Oct 8, 2021Microbiology

Restored TDCA and Valine Levels Imitate the Effects of Bariatric Surgery

S. G. Tullius, M. Quante, J. Iske, T. Heinbokel, B. N. Desai, H. R. C. Biefer, Y. Nian, F. Krenzien, H. Uehara, R. Maenosono, H. Azuma, J. Pratschke, C. Falk, T. Lo, E. Sheu, , A. Elkhal

The paper shows that restoring TDCA and valine levels in obese mice mimics the weight loss effects of bariatric surgery.

bioRxiv · Jan 11, 2021Cell Biology