Author

M. Yoda

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cancer Biology· 2 Biochemistry· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Assay:bulk RNA-seq· 3 Organism:mouse· 3 Research Focus:cell type annotation· 2 Research Focus:cell-cell communication· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Approach:statistical model· 2 Research Focus:cell signaling & trafficking· 1

Frequent Co-Authors

S. Kawaoka· 3 shared C. He· 2 shared K. Kawaguchi· 2 shared M. Takahashi· 2 shared M. Toi· 2 shared R. Konishi· 2 shared R. Mizuno· 2 shared T. Bamba· 2 shared

Activity by Year

2022-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 43 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

3 shared papersCancer Biology

S. Kawaoka

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Low albumin status accompanies multi-layered immunosuppressive phenotypes in metastatic breast cancer patients

Y. Nakamura, M. Yoda, Y. Izumi, Y. Kashima, M. Takahashi, K. Nakatani, T. Bamba, C. He, R. Konishi, D. P. Saldajeno, A. Vandenbon, Y. Suzuki, M. Toi, K. Kawaguchi, S. Kawaoka

The paper investigates the association between low albumin levels and immunosuppressive phenotypes in metastatic breast cancer patients using transcriptomic and metabolomic data.

bioRxiv · Sep 5, 2023 · PreprintCancer Biology

Serum amyloid alpha 1-2 are not required for systemic inflammation in the 4T1 murine breast cancer model

C. He, R. Konishi, A. Harata, Y. Nakamura, R. Mizuno, M. Yoda, M. Toi, K. Kawaguchi, S. Kawaoka

This study shows that SAA1-2 proteins are not essential for systemic inflammation in the 4T1 murine breast cancer model.

bioRxiv · Sep 26, 2022 · PreprintCancer Biology

Nicotinamide-N-methyltransferase is essential for SAM and 1-methylnicotinamide homeostasis in the AML12 hepatocyte cell line

M. Yoda, R. Mizuno, I. Yoshihiro, M. Takahashi, T. Bamba, S. Kawaoka

This study investigates the role of NNMT in regulating SAM and MNAM metabolism in AML12 hepatocytes, finding that NNMT is a significant consumer of SAM and critical for MNAM production.

bioRxiv · Sep 25, 2022 · PreprintBiochemistry