Author

Martin Walsh

Fox Chase Cancer Center

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Assay:DNA methylation· 2 Organism:human· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Assay:proteomics· 1

Frequent Co-Authors

Alfonso BellacosaiD· 2 shared Monika Maria TomaiD· 2 shared Tomasz SkórskiiD· 2 shared Aidan McKelvey· 1 shared Anna-Mariya Kukuyan· 1 shared Anushka Udara· 1 shared Bac Viet LeiD· 1 shared Benjamin I. FermaniD· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersCancer Biology

Alfonso BellacosaORCID

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2 papers

Recent·Most cited

DNMT1-Mediated Epigenetic Reprogramming Drives KMT2A Amplifications and Rearrangements

Madison A. Lineberger, Zach H. Gray, Benjamin I. Ferman, Erin O Donnell, Rebecca G. Smith, Kathleen L Schiela, Anushka Udara, Kashish Chetal, Chloe Azadegan, Monika Maria Toma, Ethan Sumner, John Santoro, Christopher Miranda, Manna Ahmed, Jade Wilson, , Johnathan R. Whetstine

The paper identifies a DNA-histone methylation axis that drives KMT2A amplifications and rearrangements in leukemia, revealing therapeutic targets.

bioRxiv · Sep 22, 2026 · PreprintCancer Biology

Pol is a functional dependency and therapeutic vulnerability in DNMT3A deficient leukemia cells

Bac Viet Le, Umeshkumar M. Vekariya, Monika Maria Toma, Margaret Nieborowska-Skorska, Marie‐Christine Caron, Malgorzata Gozdecka, Zayd Haydar, Martin Walsh, Jayashri Ghosh, Elaine Vaughan‐Williams, Paulina Podszywalow‐Bartnicka, Anna-Mariya Kukuyan, Sylwia Ziółkowska, Jessica Atkins, Emir Hadzijusufovic, , Tomasz Skórski

This study identifies Polθ as a therapeutic target in DNMT3A-mutated leukemia by showing its essential role in DNA repair and replication fork restart, and demonstrates that its inhibition enhances standard therapies.

Cell Reports Medicine · Mar 1, 2026Peer-reviewedCancer Biology