Author

N. Fujiwara

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Assay:proteomics· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:differential expression· 1

Frequent Co-Authors

C. Lewis· 1 shared G. Pierron· 1 shared H. Takakuwa· 1 shared H. Yoshino· 1 shared H. Zhu· 1 shared L. Li· 1 shared M. Hoare· 1 shared M. Zhu· 1 shared

Activity by Year

2023-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperCell Biology

C. Lewis

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2 papers

Recent·Most cited

Shell protein composition specified by NEAT1 domains dictates the formation of paraspeckles as distinct membraneless organelles

H. Takakuwa, T. Yamazaki, S. Souquere, S. Adachi, H. Yoshino, N. Fujiwara, T. Yamamoto, N. Tohru, S. Nakagawa, G. Pierron, T. Hirose

The paper identifies how NEAT1 domains specify shell protein composition in paraspeckles, enabling their independence from nuclear speckles through phase separation.

bioRxiv · May 21, 2023 · PreprintCell Biology

Positive selection of somatically mutated clones identifies adaptive pathways in metabolic liver disease

Z. Wang, S. Zhu, Y. Jia, Y. Wang, N. Kubota, N. Fujiwara, R. Gordillo, C. Lewis, M. Zhu, T. Sharma, L. Li, Q. Zeng, Y.-H. Lin, M.-H. Hsieh, P. Gopal, , H. Zhu

The paper identifies adaptive pathways in metabolic liver disease by selecting somatically mutated clones in mice and humans, finding mutations that ameliorate lipotoxicity and promote clonal expansion.

bioRxiv · Mar 20, 2023Cell Biology