Author

Tatjana Dorn

TUM Klinikum

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Bioengineering· 1 Developmental Biology· 1 Systems Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Organism:human· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Assay:imaging· 1

Frequent Co-Authors

Alessandra MorettiiD· 3 shared Christian KupattiD· 3 shared Achim KrügeriD· 1 shared Alexander Genzelinakh· 1 shared Alexander GoedeliD· 1 shared Andrea Baehr· 1 shared Andreas DendorferiD· 1 shared Arne SkerraiD· 1 shared

Activity by Year

2021-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 28 mutual co-author links

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3 shared papersBioengineering

Alessandra MorettiORCID

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Author profile →

3 papers

Recent·Most cited

A new bioorthogonal PET reporter gene for cardiac regenerative medicine binding to DTPA-lanthanide complexes

Sepideh Seyfi, Tatjana Dorn, Christine M Poch, Martin Grashei, Eleonore Baier, Volker Morath, Veronika Fricke, Katja Fritschle, Arne Skerra, Markus Schwaiger, Christian Kupatt, Karl-Ludwig Laugwitz, Alessandra Moretti, Wolfgang Weber

This paper develops a bioorthogonal PET reporter gene for tracking hiPSC-derived cardiac cells in vitro and ex vivo, showing it maintains function without adverse effects.

bioRxiv · Sep 28, 2026 · PreprintBioengineering

Circuit to target approach defines an autocrine myofibroblast loop that drives cardiac fibrosis

Shoval Miyara, Miri Adler, Kfir Baruch Umansky, Daniel Häußler, Elad Bassat, Yalin Divinsky, Jacob Elkahal, David Kain, Daria Lendengolts, Ricardo O. Ramirez Flores, Hanna Bueno-Levy, Ofra Golani, Tali Shalit, Michael Gershovits, Eviatar Weizman, , Eldad Tzahor

The paper models a macrophage-myofibroblast circuit to predict fibrosis types and shows targeting the myofibroblast autocrine loop reduces fibrosis after myocardial infarction.

Cell Systems · Feb 18, 2025Peer-reviewedSystems Biology

Sequential defects in cardiac lineage commitment and maturation cause hypoplastic left heart syndrome

Markus Krane, Martina Dreßen, Gianluca Santamaria, Ilaria My, Christine M. Schneider, Tatjana Dorn, Svenja Laue, Elisa Mastantuono, Riccardo Berutti, Hilansi Rawat, Ralf Gilsbach, Pedro Schneider, Harald Lahm, Sascha Schwarz, Stefanie A. Doppler, , Alessandra Moretti

This study identifies sequential defects in cardiac lineage commitment and maturation in hypoplastic left heart syndrome using multi-omics approaches and iPSC modeling.

Circulation · Oct 25, 2021Peer-reviewedDevelopmental Biology