Author

V. P. Reddy

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 1 Cancer Biology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:drug response / drug discovery· 1 Organism:human· 1 Organism:mouse· 1

Frequent Co-Authors

J. N. Glasgow· 2 shared R. R. Sevalkar· 2 shared A. Agarwal· 1 shared A. Farooqi· 1 shared A. Hindle· 1 shared A. J. C. Steyn· 1 shared A. J. Steyn· 1 shared A. Smith· 1 shared

Activity by Year

2021-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 18 mutual co-author links

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2 shared papersMicrobiology

J. N. Glasgow

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

NAD(H) homeostasis is essential for host protection mediated by glycolytic myeloid cells in tuberculosis

H. T. Pacl, K. C. Chinta, V. P. Reddy, S. Nadeem, R. R. Sevalkar, K. Nargan, K. Lumamba, T. Naidoo, J. N. Glasgow, A. Agarwal, A. J. C. Steyn

This study shows that NAD(H) homeostasis in glycolytic myeloid cells is critical for host protection against tuberculosis, with nicotinamide reducing Mtb lung burden.

bioRxiv · Sep 11, 2022 · PreprintMicrobiology

Mycobacterium tuberculosis DosS binds H2S through its Fe3+ heme iron to regulate the Dos dormancy regulon

R. R. Sevalkar, J. N. Glasgow, M. Pettinati, M. A. Martin, V. P. Reddy, S. Basu, E. Alipour, D. B. Kim-Shapiro, D. A. Estrin, J. R. Lancaster, A. J. Steyn

This study shows Mycobacterium tuberculosis uses its DosS sensor kinase to detect H2S via Fe3+ heme binding, activating the dormancy regulon.

bioRxiv · Jun 21, 2021 · PreprintBiochemistry

ALT Neuroblastoma Chemoresistance due to ATM Activation by Telomere Dysfunction is Reversible with the ATM Inhibitor AZD0156

B. Koneru, A. Farooqi, T. H. Nguyen, W. H. Chen, A. Hindle, C. Eslinger, M. R. Makena, T. A. Burrow, J. Wilson, A. Smith, V. P. Reddy, E. Cadogan, S. T. Durant, C. P. Reynolds

This study shows that ATM activation due to telomere dysfunction causes chemoresistance in ALT neuroblastoma, which can be reversed by the ATM inhibitor AZD0156.

bioRxiv · Apr 6, 2021Cancer Biology