Author

V. Servellita

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 2 Organism:human· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:ecology & biodiversity· 1 Assay:metabolomics· 1 Assay:metagenomics· 1 Organism:mouse· 1

Frequent Co-Authors

C. Y. Chiu· 3 shared A. Sotomayor-Gonzalez· 2 shared A. Rustagi· 1 shared B. Castillo-Rojas· 1 shared C. A. Blish· 1 shared C. Hanson· 1 shared C. J. Miller· 1 shared C. K. Yinda· 1 shared

Activity by Year

2021-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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3 shared papersMicrobiology

C. Y. Chiu

Mutual co-authors in cluster

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3 papers

Recent·Most cited

Longitudinal comparison of the developing gut virome in infants and their mothers

S. Federman, Y. Santos, T. Haggerty, A. Sotomayor-Gonzalez, V. Servellita, R. E. Ley, J. Parsonnet, C. Y. Chiu

This study compares the gut virome of 53 infants and their mothers over three years, finding distinct viral compositions and ongoing maturation of the infant virome.

bioRxiv · May 13, 2022 · PreprintMicrobiology

SARS-CoV-2 Spike triggers barrier dysfunction and vascular leak via integrins and TGF-β signaling

S. B. Biering, F. Tramontini Gomes de Sousa, L. V. Tjang, F. Pahmeier, R. Ruan, S. F. Blanc, T. S. Patel, C. M. Worthington, D. R. Glasner, B. Castillo-Rojas, V. Servellita, N. T. Lo, M. P. Wong, C. M. Warnes, D. R. Sandoval, , E. Harris

This study shows SARS-CoV-2 Spike protein causes barrier dysfunction and vascular leak through integrins and TGF-β signaling, independent of viral replication and ACE2.

bioRxiv · Dec 10, 2021 · PreprintMicrobiology

The B.1.427/1.429 (epsilon) SARS-CoV-2 variants are more virulent than ancestral B.1 (614G) in Syrian hamsters

T. Carroll, D. Fox, N. van Doremalen, E. Ball, M. K. Morris, A. Sotomayor-Gonzalez, V. Servellita, A. Rustagi, C. K. Yinda, L. Fritts, J. R. Port, Z.-M. Ma, M. Holbrook, J. Schulz, C. A. Blish, , C. J. Miller

This study shows that the B.1.427/1.429 (epsilon) SARS-CoV-2 variants cause more severe disease and higher viral loads in the upper respiratory tract in Syrian hamsters compared to the ancestral B.1 (614G) strain.

bioRxiv · Aug 25, 2021Microbiology