Author

Wensheng Tian

University of Ottawa

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Molecular Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Research Focus:protein structure prediction· 1 Assay:proteomics· 1

Frequent Co-Authors

Ryan Charles RusselliD· 2 shared Anne Jackson· 1 shared Ariel MadrigaliD· 1 shared Bürge UlukaniD· 1 shared C.N.R. RaoiD· 1 shared Campbell McInnesiD· 1 shared David Chaim RubinszteiniD· 1 shared Elizabeth Hill· 1 shared

Activity by Year

2019-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 31 mutual co-author links

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2 shared papersCancer Biology

Ryan Charles RussellORCID

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2 papers

Recent·Most cited

Lysyl oxidase drives ccRCC progression by coordinating HIF-2α transcription program with tumor microenvironment

Bürge Ulukan, Özge Saatci, Ariel Madrigal, Minjun Kim, Wensheng Tian, Mustafa Soytaş, Zohreh Mehrjoo, Ozlem Sener Sahin, Kukkamadi Sreenivas, C.N.R. Rao, Tamiko Nishimura, Virginie Pillon, Jean-Sebastian Anoma, Elizabeth Hill, Janusz Rak, , Ozgur Sahin

This study identifies LOX as a driver of ccRCC progression by sustaining HIF-2α transcription and modulating the tumor microenvironment.

bioRxiv · Jul 27, 2026 · PreprintCancer Biology

ULK-mediated phosphorylation of ATG16L1 promotes xenophagy, but destabilizes the ATG16L1 Crohn’s mutant

Reham M. Alsaadi, Truc T. Losier, Wensheng Tian, Anne Jackson, Zhihao Guo, David Chaim Rubinsztein, Ryan Charles Russell

This study shows ULK1 phosphorylates ATG16L1, enhancing wildtype function but destabilizing the Crohn’s disease-associated mutant, thereby affecting xenophagy.

EMBO Reports · May 24, 2019Peer-reviewedMolecular Biology