Author

Yinghui Sui

University of California San Diego

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Developmental Biology· 1 Genomics· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Research Focus:gene regulatory network inference· 3 Approach:statistical model· 3 Research Focus:differential expression· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Approach:experimental assay· 1 Research Focus:GWAS / association mapping· 1

Frequent Co-Authors

Chun ZengiD· 3 shared Maike SanderiD· 3 shared Jinzhao WangiD· 2 shared Joshua ChiouiD· 2 shared Allen WangiD· 1 shared Allison Deogaygay· 1 shared Andrea C. Carrano· 1 shared Austin R. Harrington· 1 shared

Activity by Year

2019-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 32 mutual co-author links

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3 shared papersPhysiology

Chun ZengORCID

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Author profile →

3 papers

Recent·Most cited

Workload-induced changes to cell state contribute to β-cell failure in diabetes

Somesh Sai, Fenfen Liu, Austin R. Harrington, Han Zhu, Ibrahim Omar, Chun Zeng, Medhavi Mallick, Yinghui Sui, Maike Sander, Matthew Wortham

This study shows workload-induced β-cell overstimulation leads to failure in T2D, identifying a compensating transcriptional state and the role of Lsd1 in restraining state transitions.

bioRxiv · May 18, 2026 · PreprintPhysiology

Single cell chromatin accessibility reveals pancreatic islet cell type- and state-specific regulatory programs of diabetes risk

Joshua Chiou, Chun Zeng, Cheng Zhang, Jee Yun Han, Michael Schlichting, Michael Miller, Robert Mendez, Serina Huang, Jinzhao Wang, Yinghui Sui, Allison Deogaygay, Mei-Lin Okino, Yunjiang Qiu, Ying Sun, Parul Kudtarkar, , Kyle J. Gaulton

The paper uses single-cell chromatin accessibility to identify cell type- and state-specific regulatory programs associated with type 2 diabetes risk.

Nature Genetics · Apr 1, 2021Peer-reviewedGenomics

A network of microRNAs acts to promote cell cycle exit and differentiation of human pancreatic endocrine cells

Wen Long Jin, Francesca Mulas, Bjoern Gaertner, Yinghui Sui, Jinzhao Wang, Ileana Matta, Chun Zeng, Nicholas K. Vinckier, Allen Wang, Kim-Vy Nguyen-Ngoc, Joshua Chiou, Klaus H. Kaestner, Kelly A. Frazer, Andrea C. Carrano, Hung-Ping Shih, Maike Sander

The paper identifies miRNAs that promote cell cycle exit and differentiation of human pancreatic endocrine cells through regulatory networks.

iScience · Nov 1, 2019Peer-reviewedDevelopmental Biology