James A. Walker

The paper uses high-content microscopy and machine learning to identify a cell morphology signature of NF1 genotype in Schwann cells, but model performance declines when applied to new cell lines.

This study uses Drosophila to model insomnia-associated genes and identifies genetic factors influencing sleep quantity, timing, and quality, revealing subtype-specific effects.

This study shows that dNf1 in Drosophila regulates metabolic rate through neuronal mechanisms, with effects on feeding, lipid stores, and energy expenditure.

The paper shows HTT represses ABL activity, which is necessary for axonal growth in Drosophila mushroom bodies.

This study identifies peripheral chemosensory neuron dysfunction in Drosophila as a cause of social behavioral deficits linked to NF1, showing that Nf1 regulates Ras signaling in ppk23+ neurons to maintain normal social behavior.

The paper describes a gene-drive system in Drosophila controlled by an engineered Cas9 and a synthetic small molecule to enable chemical control of inheritance.