Author

Maria C. Panepinto

Cold Spring Harbor LaboratoryORCID 0000-0001-7040-8181Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Assay:ChIP-seq· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

Paolo CifaniiD· 3 shared David L. SpectoriD· 2 shared Aktan AlpsoyiD· 1 shared Amanda M. CafieroiD· 1 shared Anand SwaroopiD· 1 shared Anjani Kumari· 1 shared Christopher R. VakociD· 1 shared Christy Felice· 1 shared

Activity by Year

2024-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 23 mutual co-author links

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3 shared papersCancer Biology

Paolo CifaniORCID

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3 papers

Recent·Most cited

Cancer BiologybioRxivPreprintOct 28, 2026

PTP1B supports survival and regulates CFL1 and HSP90a abundance in trastuzumab-resistant HER2-positive breast cancer.

Vinay Mandati, Maria C. Panepinto, Christy Felice…·Nicholas K. Tonks

This study identifies PTP1B as a survival dependency in trastuzumab-resistant HER2-positive breast cancer, showing it regulates CFL1 and HSP90a abundance through dephosphorylation.

DOI 10.64898/2026.10.23.761095

humanexperimental assay

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Molecular BiologybioRxivPreprintApr 17, 2025

Aerobic glycolysis and lactate regulate histone H3K18Lactylation occupancy to fine-tune gene expression in developing and mature retina

Mohita Gaur, Xulong Liang, Matthew J. Brooks…·Anand Swaroop

The paper shows that glycolysis and lactate regulate histone H3K18 lactylation to control gene expression in retinal development and maturation.

2 citations

mouseChIP-seq

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Cancer BiologyCancer DiscoveryPeer-reviewedJul 25, 2024

MARK2/MARK3 kinases are catalytic co-dependencies of YAP/TAZ in human cancer

Olaf Klingbeil, Damianos S. Skopelitis, Claudia Tonelli…·Christopher R. Vakoc

This study identifies MARK2/3 as essential kinases for YAP/TAZ function in cancer and shows their inhibition can restore Hippo pathway tumor suppression.

31 citationstop 4%

humanproteomics

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