Kirill E. Volynski

The paper identifies a minimal set of presynaptic proteins that regulate different modes of Ca2+-triggered vesicle fusion and short-term plasticity.

The paper quantifies asynchronous glutamate exocytosis in synapses, showing it is enhanced in low release probability synapses and more widely dispersed than synchronous release.

This study shows that Syt1 oligomerisation is essential for clamping spontaneous and asynchronous neurotransmitter release, revealing a mechanism for regulating release modes.

This study identifies a Vamp2 mutation in mice that causes sleep-wake transition deficits by reducing synaptic release probability.

The paper demonstrates that mossy fibers count action potentials to transmit information to CA3 pyramidal neurons through presynaptic calcium dynamics and vesicle replenishment.