Jens Januschke

The paper shows that cell size can override fate determinants in Drosophila neuroblasts, with larger daughters retaining neuroblast-like properties despite inheriting GMC determinants.

The paper shows that Erv14/cornichon receptor dynamics and bilayer thickness influence directional ER-Golgi traffic through a mechanism called HEIST.

This study identifies RhoGAP54D as a regulator of Myosin dynamics in Drosophila neuroblasts, showing it suppresses Myosin activity during interphase and metaphase and initiates directional Myosin flow during anaphase to promote cell size asymmetry.

The paper uses an active matter model to simulate and reproduce actomyosin oscillations in Drosophila cells during morphogenesis, showing that cell geometry and polarity drive these dynamics.

This study shows that CDK1 phosphorylates PAR-3 in asymmetrically dividing cells, affecting polarity and fate determination in Drosophila neuroblasts and sensory organ precursors.

The paper develops fluorescent reporter tools in human pluripotent stem cells to study neural development through live imaging of cell behaviors.

This study shows that rapid aPKC inactivation in Drosophila follicular epithelium causes tissue rupture through increased apical constriction, not polarity loss.

The paper identifies that Drosophila neural stem cells use the position of their last-born daughter cell as an extrinsic cue to orient their division.