Author

Erika Bereczki

Karolinska InstitutetORCID 0000-0002-0373-7417Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 1 Research Focus:differential expression· 1 Organism:Drosophila· 1 Approach:experimental assay· 1 Organism:mouse· 1 Approach:statistical model· 1

Frequent Co-Authors

Benjamin KottleriD· 1 shared Catarina Moreira Pinho· 1 shared Christopher J. H. ElliottiD· 1 shared Dag AarslandiD· 1 shared Erik Berger· 1 shared Frank HirthiD· 1 shared Giacomo DentoniiD· 1 shared Gonçalo M. PoçasiD· 1 shared

Activity by Year

2021-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperNeuroscience

Benjamin KottlerORCID

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2 papers

Recent·Most cited

NeurosciencebioRxivPreprintMar 7, 2023

Early mitochondrial dysfunction proceeds neuroinflammation, synaptic alteration, and autophagy impairment in hippocampus of App knock-in Alzheimer mouse models

Luana Naia, Makoto Shimozawa, Erika Bereczki…·Per Nilsson

This study identifies early mitochondrial dysfunction, neuroinflammation, and autophagy impairment in App knock-in Alzheimer's mouse models through time-course transcriptome analysis.

2 citations

mousebulk RNA-seq

𝕏 PostBluesky

NeuroscienceBrain CommunicationsPeer-reviewedMar 19, 2021

Presynaptic accumulation of α-synuclein causes synaptopathy and progressive neurodegeneration

Jéssika Cristina Bridi, Erika Bereczki, Saffron K. Smith…·Frank Hirth

This study shows that α-synuclein accumulation in presynaptic terminals causes synaptic dysfunction and progressive neurodegeneration in a Drosophila model, with similar findings in patient samples.

24 citations

Drosophilaexperimental assay

𝕏 PostBluesky