Author

Ilana Buchumenski

Bar-Ilan UniversityORCID 0000-0002-1333-3969Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

Erez Y. LevanoniD· 2 shared Aaron Orgel· 1 shared Alyssa R. Fishbein· 1 shared Anton Davydenko· 1 shared Ariel FeigliniD· 1 shared Diego Capcha-RodrigueziD· 1 shared Dror SharoniD· 1 shared Eyal BaniniD· 1 shared

Activity by Year

2024-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersBiochemistry

Erez Y. LevanonORCID

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2 papers

Recent·Most cited

PACT prevents aberrant activation of PKR by endogenous dsRNA without sequestration

Sadeem Ahmad, Tao Zou, Jihee Hwang, Linlin Zhao, Xi Wang, Anton Davydenko, Ilana Buchumenski, Patrick Zhuang, Alyssa R. Fishbein, Diego Capcha-Rodriguez, Aaron Orgel, Erez Y. Levanon, Sua Myong, James Jeiwen Chou, Matthew M. Meyerson, Sun Hur

This study shows PACT inhibits PKR activation by endogenous dsRNA through restricting PKR movement on dsRNA, without sequestering dsRNA, thereby adjusting the activation threshold for long dsRNA.

Nature Communications · Apr 8, 2025Peer-reviewedBiochemistry

The prevalent NR2E3 c.932G>A mutation induces aberrant splicing that can be rescued using splice-shifting antisense oligonucleotides

Yehezkel Sztainberg, Maya David Teitelbaum, Ilana Buchumenski, Hagit T. Porath, Dror Sharon, Eyal Banin, Rotem Karni, Erez Y. Levanon, Ariel Feiglin

This study identifies aberrant splicing caused by the NR2E3 c.932G>A mutation and shows that splice-shifting ASOs can restore full-length protein function.

bioRxiv · May 1, 2024 · PreprintGenomics