Kaoutsar Nasrallah

This study investigates the role of GluN2D-containing NMDA receptors in dentate granule cells, showing their importance in excitability, synaptic plasticity, and spatial memory.

This study shows that loss of MEF2C in embryonic cortical GABAergic interneurons reduces their number and maturation, leading to altered brain function and neurodevelopmental disorder-like behaviors.

This study shows that dopamine D2 receptors in mossy cells reduce excitatory transmission and are essential for hippocampal function, with implications for anxiety and epilepsy.

This study investigates NMDA receptor plasticity at different synapses in hippocampal dentate granule cells, finding that MPP-GC synapses exhibit homosynaptic LTP while inducing heterosynaptic LTP at LPP-GC synapses.

This paper shows BDNF induces its own release to mediate long-lasting increase in neurotransmitter release through TrkB and calcium-dependent mechanisms.

This study identifies adenosine/A2A receptor retrograde signaling as a mechanism for presynaptic long-term potentiation and seizure promotion in hippocampal circuits.

The paper shows that initial seizures in mice increase mossy cell-granule cell synaptic transmission via BDNF-dependent LTP, which worsens seizures and suggests a mechanism for epileptogenesis.