Renata Batista‐Brito

The paper shows that sensory adaptation and expectation-related signals in visual cortex are represented as distinct population dimensions, with ACC input modulating the expectation-related axis.

This study identifies Sst-Chodl inhibitory neurons that promote cortical synchrony and sleep by exerting long-range inhibition during low-arousal states.

The paper isolates individual cycles of neural oscillations from spiking activity in mice, revealing time-scale-dependent correlations between brain regions and improving trial alignment in sensory experiments.

The paper identifies enhancer-AAV tools for specific neuronal subtypes and validates them across species.

This paper introduces gcPCA, a hyperparameter-free method for comparing high-dimensional datasets, demonstrating its utility in detecting hippocampal replay and type II diabetes heterogeneity.

This study shows that loss of MEF2C in embryonic cortical GABAergic interneurons reduces their number and maturation, leading to altered brain function and neurodevelopmental disorder-like behaviors.

This study disrupts cholinergic retinal waves in mice and shows impaired visual cortex development and function, including altered retinotopy and sensory processing.

This study investigates the role of Sox6 in maintaining somatostatin interneuron subtype identity during migration, finding that its removal reduces subtype diversity in the mature cortex.

This study shows that loss of ErbB4 in PV interneurons during embryonic development disrupts cortical circuit dynamics and neuronal connectivity.

This paper demonstrates an in vivo platform for transplanting neocortical tissue prototypes that can differentiate into functional neurons and become vascularized.

The paper identifies off-manifold coding in visual cortex during sleep, showing that stimuli are encoded in a subspace distinct from movement-related activity.

The paper identifies enhancers for targeting specific neuronal subtypes across species and demonstrates their utility in circuit manipulation.

The paper investigates the role of three GABAergic interneuron populations in a mouse model of Rett Syndrome, finding distinct contributions to seizure, motor, and social behavior deficits.

The paper uses single-cell RNA-seq to study the development of cortical interneurons, identifying key transcription factors and cell states involved in their diversification.