Author

Peter Burgers

Washington University in St. LouisORCID 0000-0002-3530-7235Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Biochemistry· 2 Genetics· 1

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 2 Research Focus:chromatin & epigenetic regulation· 2 Approach:experimental assay· 2 Organism:yeast· 2 Approach:biophysical modeling· 1 Research Focus:differential expression· 1 Assay:DNA methylation· 1 Research Focus:protein folding & dynamics· 1

Frequent Co-Authors

Carrie M. W. Stith· 2 shared Aditya MojumdariD· 1 shared Ayush MistryiD· 1 shared Barry S. TayloriD· 1 shared Dinshaw J. PateliD· 1 shared Edwin AntonyiD· 1 shared Fiorella GhisaysiD· 1 shared Jennifer A. CobbiD· 1 shared

Activity by Year

2020-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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2 shared papersBiochemistry

Carrie M. W. Stith

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3 papers

Recent·Most cited

Opposing regulation by Rev1 of DNA polymerase zeta activity on damaged versus undamaged DNA

Rachel Bezalel‐Buch, Carrie M. W. Stith, Аlena V. Makarova, Sara K. Binz, Peter Burgers

This study shows Rev1 inhibits Pol zeta activity on undamaged DNA but stimulates it at damaged sites, with the N-terminal M1 motif mediating this regulation.

Nucleic Acids Research · May 27, 2026Peer-reviewedBiochemistry

Dissecting the role of PCNA and Pif1 in replication of individual DNA molecules by DNA polymerase δ.

Manal S. Zaher, Carrie M. W. Stith, Ayush Mistry, Purna Bahadur Poudel, Edwin Antony, Peter Burgers, Roberto Galletto

This study uses single-molecule techniques to show PCNA, Pol δ, and Pif1 collaborate in DNA replication, with PCNA stabilization and diffusion reduction by both Pol δ and Pif1.

bioRxiv · May 21, 2026 · PreprintBiochemistry

Modeling Cancer Genomic Data in Yeast Reveals Selection Against ATM Function During Tumorigenesis

Marcel Hohl, Aditya Mojumdar, Sarem Hailemariam, Vitaly Kuryavyi, Fiorella Ghisays, Kyle Sorenson, Matthew Chang, Barry S. Taylor, Dinshaw J. Patel, Peter Burgers, Jennifer A. Cobb, John H.J. Petrini

The paper models mutations in the Mre11 complex in yeast to show that defects in ATM signaling, rather than DNA repair, are more critical for tumor suppression.

PLoS Genetics · Mar 18, 2020Peer-reviewedGenetics