Author

María Antonia Odena

Institute for Research in BiomedicineORCID 0000-0002-4530-7829Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Bioinformatics· 1 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:benchmarking· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Organism:mouse· 1 Assay:proteomics· 1 Research Focus:software / tool· 1 Approach:statistical model· 1

Frequent Co-Authors

Camille Stephan‐Otto AttoliniiD· 2 shared Ignasi Folch-I-CasanovasiD· 2 shared Alba Duran-Cortines· 1 shared Alberto MartíniD· 1 shared Alberto VillanuevaiD· 1 shared Almudena Escobar-NiñoiD· 1 shared Ana Montero‐CalleiD· 1 shared Ángela Peralbo‐MolinaiD· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 30 mutual co-author links

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2 shared papersBioinformatics

Camille Stephan‐Otto AttoliniORCID

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2 papers

Recent·Most cited

A generalizable normalization framework to decouple protocol and instrument effects: Application to high-sensitivity proteomics multicentric study (PME13)

Gianluca Arauz‐Garofalo, Sergio Ciordia, Anne Gonzalez de Peredo, Karima Chaoui, Jeewan Babu Rijal, Valeriane Gaxotte, Ignasi Folch-I-Casanovas, Mikel Azkargorta, Ruben Almey, Kerman Aloria, Busra Aytul Kirim, Rodrigo Barderas, Sophie Braga-Lagache, Enrique Calvo, Eduardo Chicano‐Gálvez, , Fernando José Corrales

This paper introduces a normalization framework to decouple protocol and instrument effects in multicenter proteomics studies, demonstrating its application to the PME13 dataset.

bioRxiv · Aug 20, 2026 · PreprintBioinformatics

Targeted KRAS(G12V) degradation elicits efficient and durable lung adenocarcinoma regression in vivo

Alberto Martín, Inés María García-Pérez, Sonia San José, Pep Rojo, Carlos Riego‐Mejías, Cristina Teodósio, Bárbara M. G. Barbosa, Carolina Sánchez, Ignasi Folch-I-Casanovas, María Antonia Odena, Sònia Jarió, Sara Hijazo‐Pechero, Silvia M. Rodríguez-López, Rodrigo Entrialgo‐Cadierno, Marie‐Julie Nokin, , Cristina Mayor‐Ruiz

This study shows that targeted KRAS(G12V) degradation causes durable lung adenocarcinoma regression in mice, with tumor relapse linked to proteolysis machinery dysregulation.

Cancer Research · May 27, 2026Peer-reviewedCancer Biology