Author

Tripti Nair

University of Southern CaliforniaORCID 0000-0002-5380-4622Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cell Biology· 2 Genetics· 1

Signature Assays, Organisms & Methods

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Organism:C. elegans· 3 Approach:statistical model· 3 Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Research Focus:cell-cell communication· 2 Research Focus:differential expression· 2 Research Focus:gene regulatory network inference· 2 Approach:benchmark study· 1

Frequent Co-Authors

Sean P. CurraniD· 3 shared Aeowynn J. CoakleyiD· 2 shared Athena AlcalaiD· 2 shared Bérénice Anath BenayouniD· 2 shared Darius Moaddeli· 2 shared Gilberto GarciaiD· 2 shared Maxim AverbukhiD· 2 shared Naibedya Dutta· 2 shared

Activity by Year

2024-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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3 shared papersCell Biology

Sean P. CurranORCID

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3 papers

Recent·Most cited

Form and function of actin impacts actin health and aging.

Maxim Averbukh, Hunter T. Nelson, Tiffany V. Wang, Caitlin M. Lange, Athena Alcala, Juri Kim, Daniella Suh, Matthew Vega, Shihong Max Gao, Naibedya Dutta, Khristina E. Young, Rebecca Aviles Barahona, Aeowynn J. Coakley, Joe A. Gerke, Sofia F. Odron, , Ryo Higuchi‐Sanabria

This study investigates how actin structure and function affect aging in C. elegans and finds that actin dysfunction accelerates aging hallmarks and correlates with human age-related decline in gait speed.

iScience · Aug 24, 2026Peer-reviewedCell Biology

Distinct mechanisms of non-autonomous UPRER mediated by GABAergic, glutamatergic, and octopaminergic neurons.

Aeowynn J. Coakley, Adam J. Hruby, Jing Wang, Andrew Bong, Tripti Nair, Carmen M. Ramos, Athena Alcala, Daniel Hicks, Maxim Averbukh, Naibedya Dutta, Darius Moaddeli, Camilla Pearson, Cynthia J. Siebrand, Mattias de los Rios Rogers, Arushi Sahay, , Ryo Higuchi‐Sanabria

The paper identifies distinct roles of GABAergic, glutamatergic, and octopaminergic neurons in mediating non-autonomous UPRER through transcriptomic and functional analyses in C. elegans.

Communications Biology · Nov 24, 2025Peer-reviewedCell Biology

Serotonin deficiency from constitutive SKN-1 activation drives pathogen apathy.

Tripti Nair, Brandy A. Weathers, Nicole L. Stuhr, James D. Nhan, Sean P. Curran

The paper shows that constitutive SKN-1 activation in C. elegans leads to serotonin deficiency, causing apathy toward pathogens and reduced survival.

Nature Communications · Sep 16, 2024Peer-reviewedGenetics