Ramón Lorenzo-Redondo

This study identifies TMEM106B as an alternative receptor for SARS-CoV-2 S-E484D variant entry, independent of ACE2, in airway-derived cell models.

This study identifies a tissue virus microenvironment with stress responses that supports SIV persistence, contrasting with transient reservoirs that show immune activity.

This study uses spatial transcriptomics to compare HIV/SIV reservoirs with tumor microenvironments, identifying shared gene pathways and potential therapeutic targets.

This study identifies loss of intestinal 1,2-fucose in HIV-treated individuals as a modifiable host-microbiome mechanism linked to inflammation, barrier dysfunction, and aging.

This study investigates RSV clades associated with prophylactic breakthrough infections in pediatric transmission clusters and finds that clade associations are driven by transmission dynamics rather than resistance.

This study shows that MIF overexpression after SARS-CoV-2 infection promotes neural regeneration in human brain organoids through EGFR signaling and self-upregulation.

This study develops microglia-containing cerebral organoids (CO-iMs) to model HIV-1 infection and its effects on neuroinflammation and neuronal function.

This paper shows that inhibiting the Super Elongation Complex (SEC) with KL-2 increases HIV-1 transcription and reactivates latent virus, especially when combined with other latency reversing agents.

The paper examines alveolar T cell responses in SARS-CoV-2 pneumonia patients, finding that early interferon signaling and targeting of structural proteins correlate with better clinical outcomes.