Author

Hans Georg Richter

Austral University of ChileORCID 0000-0002-7224-9388Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 2 Organism:human· 2 Organism:mouse· 2 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Assay:proteomics· 1 Approach:statistical model· 1

Frequent Co-Authors

Alicia M. KembleiD· 1 shared Arun ThiruvalluvaniD· 1 shared Benoit Hornsperger· 1 shared Bernd KuhniD· 1 shared Brad J. Geddes· 1 shared Britta EngelhardtiD· 1 shared Carlos SpichigeriD· 1 shared Christian Enrique González-BillaultiD· 1 shared

Activity by Year

2020-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperNeuroscience

Alicia M. KembleORCID

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

A potent and selective inhibitor for the modulation of MAGL activity in the neurovasculature

Alicia M. Kemble, Benoit Hornsperger, Iris Ruf, Hans Georg Richter, Jörg Benz, Bernd Kuhn, Dominik Heer, Matthias Wittwer, Britta Engelhardt, Uwe Grether, Ludovic Collin

This study characterizes a novel MAGL inhibitor in a mouse model and human cells, showing its expression in neurovascular unit cells but no effect on blood-brain barrier permeability or cytokine production.

PLoS ONE · Sep 9, 2022Peer-reviewedNeuroscience

Insulin-like growth factor 2 (IGF2) protects against Huntington's disease through the extracellular disposal of protein aggregates

Paula García-Huerta, Paulina Troncoso-Escudero, Di Wu, Arun Thiruvalluvan, Marisol Cisternas-Olmedo, Daniel R. Henríquez, Lars Plate, Pedro Chaná‐Cuevas, Cristian Saquel, Peter Thielen, Kenneth A. Longo, Brad J. Geddes, Gerardo Zelmar Lederkremer, Neeraj Sharma, Marina Shenkman, , Claudio A. Hetz

This study identifies IGF2 as a disease modifier that reduces intracellular protein aggregates by promoting their extracellular disposal in Huntington's disease models.

Acta Neuropathologica · Jul 8, 2020Peer-reviewedNeuroscience