Author

Carla M. C. Abreu

i3S - Instituto de Investigação e Inovação em Saúde, Universidade do PortoORCID 0000-0002-8070-8388Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cell Biology· 2 Genetics· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:cell signaling & trafficking· 2 Research Focus:cell type annotation· 2 Organism:C. elegans· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:clustering· 1 Research Focus:development & morphogenesis· 1 Approach:genetic perturbation / CRISPR· 1

Frequent Co-Authors

Reto GassmanniD· 2 shared Tiago J. DantasiD· 2 shared Ana R. G. De-CastroiD· 1 shared Ana Xavier CarvalhoiD· 1 shared Andrew P. CarteriD· 1 shared Erwin J.G. PetermaniD· 1 shared Guus H. HaasnootiD· 1 shared Ignasi RoigiD· 1 shared

Activity by Year

2017-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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2 shared papersCell Biology

Reto GassmannORCID

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Author profile →

3 papers

Recent·Most cited

Removal of the Ciliary Gate Allows Axoneme Extension in the Absence of Retrograde IFT

Ana R. G. De-Castro, Maria J. G. De-Castro, Guus H. Haasnoot, Reto Gassmann, Erwin J.G. Peterman, Tiago J. Dantas, Carla M. C. Abreu

This study shows that removing the ciliary gate allows axoneme extension in IFT-A-deficient cilia by enabling passive diffusion and EV release of stalled IFT machinery.

bioRxiv · Oct 21, 2025 · PreprintCell Biology

CDR2 is a dynein adaptor recruited by kinectin to regulate ER sheet organization

Vanessa Teixeira, Kashish Singh, José B. Gama, Matilde Moreira, Ricardo Celestino, Ana Xavier Carvalho, Paulo S. Pereira, Carla M. C. Abreu, Tiago J. Dantas, Andrew P. Carter, Reto Gassmann

This study identifies CDR2 as a dynein adaptor recruited by kinectin to regulate ER sheet organization and shows its role in centrosomal clustering and ER sheet stacking.

The Journal of Cell Biology · Jun 6, 2025Peer-reviewedCell Biology

Shu complex SWS1-SWSAP1 is required for mouse meiotic recombination in concert with the BRCA2 C terminus

Carla M. C. Abreu, Rohit Prakash, Peter J. Romanienko, Ignasi Roig, Scott Keeney, Maria Jasin

This study shows that the mouse SWS1-SWSAP1 complex is essential for meiotic recombination by promoting RAD51 and DMC1 focus formation, with BRCA2 C terminus playing a modulatory role.

bioRxiv · Dec 22, 2017 · PreprintGenetics