Author

Jui‐Heng Tseng

University of North Carolina at Chapel HillORCID 0000-0002-9036-9121Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Neuroscience· 2

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Organism:human· 2 Research Focus:benchmarking· 1 Assay:bulk RNA-seq· 1 Research Focus:cell-cell communication· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1

Frequent Co-Authors

A. Eden CruikshankiD· 1 shared Baggio EvangelistaiD· 1 shared Ben A. BahriD· 1 shared Carli K. OplandiD· 1 shared Diamond P. King· 1 shared Douglas H. PhanstieliD· 1 shared Ethan Paulakonis· 1 shared Hanna TrzeciakiewicziD· 1 shared

Activity by Year

2022-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperNeuroscience

A. Eden CruikshankORCID

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Synergistic MAPT mutations as a platform to uncover modifiers of tau pathogenesis

Miles R. Bryan, Michael Almeida, Kyle Pellegrino, Carli K. Opland, Ethan Paulakonis, Jake McGillion-Moore, Hanna Trzeciakiewicz, Diamond P. King, Tian Xu, Jui‐Heng Tseng, Jonathan Cummings Schisler, Nicholas G Brown, Ben A. Bahr, Todd J Cohen

This study uses synergistic MAPT mutations to create a model for tau aggregation, identifying Hsp70 as a key driver and showing its impact on neuronal function.

bioRxiv · Feb 7, 2025 · PreprintNeuroscience

Synthetic amyloid beta does not induce a robust transcriptional response in innate immune cell culture systems

Ivana Yoseli Quiroga, A. Eden Cruikshank, Kathleen S Metz Reed, Marielle L. Bond, Baggio Evangelista, Jui‐Heng Tseng, Joey V. Ragusa, Rick B. Meeker, Hyejung Won, Sarah Cohen, T.J. Cohen, Douglas H. Phanstiel

The paper assesses the transcriptional response of in vitro innate immune cell models to synthetic amyloid beta and finds no robust response, but identifies LPS and INFγ as better activators of microglia-like cells.

Journal of Neuroinflammation · Apr 23, 2022Peer-reviewedNeuroscience