Björn F.C. Kafsack

This study identifies a valine-to-leucine mutation in AP2-G that disrupts DNA binding and sexual differentiation in Plasmodium falciparum, revealing GDV1's role in ap2-g activation.

This study reveals that Plasmodium falciparum parasites can express multiple var genes or suppress var gene expression, enabling them to evade host immunity and persist in chronic infections.

This study shows that Toxoplasma gondii's PLP1 protein drives cytokine release syndrome by inducing dysregulated immune responses, leading to lethal inflammation during acute infection.

The paper shows that RNA Polymerase I inhibitors affect different Pol I-transcribed genes in Plasmodium falciparum by varying degrees, with some genes being much more sensitive than others.

The paper shows that metabolic competition for S-adenosylmethionine between histone methyltransferases and phosphoethanolamine methyltransferase regulates sexual differentiation in Plasmodium falciparum.