Benjamin A. Devlin

This study shows maternal high-fat diet causes sex-specific microglia remodeling of serotonergic reward circuits in mice, leading to altered behavior and serotonin function.

This study shows that prenatal exposure to environmental stressors disrupts gut macrophage development and causes GI dysfunction in male offspring.

The paper investigates how an influenza A-derived immune engram influences anxiety and cytokine expression through CGRP signaling in the posterior insula.

This study shows that microglial MyD88 signaling affects ECM development and interneuron maturation in the hippocampus, with sex-specific effects on PVI density and behavior.

The paper shows that Hofbauer cells and fetal brain microglia share transcriptional profiles and responses to maternal diet-induced obesity, suggesting Hofbauer cells as a noninvasive biomarker for fetal microglial programming.

This study identifies IL34 as a neuron-derived cytokine that regulates microglia maturation and synapse engulfment in the developing mouse cortex.

The paper shows maternal high-fat diet affects placenta-brain communication differently in male and female offspring, leading to sex-specific behavioral and serotonin-related changes.

The paper shows that prenatal exposure to air pollution and maternal stress in mice causes social deficits in male offspring, which can be prevented by microbiome modulation or dopamine activation.

The paper uses single-cell RNA sequencing to compare fetal brain microglia and Hofbauer cells, revealing shared transcriptional programs and sex-specific differences.

This study shows that reducing tau with a degrader prevents mitochondrial fragmentation and oxidative stress in ALS models.