Robin M. Murray

This study links genomic variation to real-time Δ9-THC response in humans, showing associations between CYP3A4 expression and psychotomimetic effects.

This study examines whether cannabis use extent predicts psychotic-like and euphoric intoxication experiences in First Episode Psychosis patients and controls, finding a significant interaction between caseness and use frequency.

The paper investigates the relationship between jumping to conclusions bias, IQ, psychosis, and polygenic risk scores in a case-control study, finding that IQ significantly predicts JTC bias while schizophrenia PRS does not.

This study identifies DNA methylation changes in blood samples from psychosis patients, including treatment-resistant schizophrenia, and links them to biological age, smoking, and cellular composition.

This study examines altered cortical gyrification in very preterm adults and its links to cognitive and mental health outcomes.

The paper examines the independent and combined effects of schizophrenia polygenic risk score and heavy cannabis use on psychotic disorder risk using EUGEI data, finding both factors independently increase risk.

This study examines the association between schizophrenia polygenic risk scores and 420 behavioural traits in the UK Biobank, identifying potential causal pathways and highlighting the need for further research.

This study identifies NT5C2 as a psychiatric risk gene, showing its role in protein translation and AMPK signaling in neural progenitor cells and Drosophila melanogaster.

This study examines the relationship between cannabis use and symptom profiles in first episode psychosis patients and population controls, finding that cannabis use correlates with positive symptoms and negative symptoms in patients.

Cross-disorder meta-analysis of eight psychiatric GWAS (232,964 cases, 494,162 controls) reports 109 loci shared by two or more disorders and three groups of correlated disorders.

This study assesses psychiatric symptomatology in adults born very preterm using a dimensional approach and finds elevated symptoms and a non-specific psychopathology profile.

This study finds that perinatal brain injury in very preterm humans is linked to reduced dopamine synthesis and hippocampal volume in adulthood.

This study identifies volumetric grey matter changes in very preterm individuals from adolescence to adulthood, showing accelerated brain maturation and regional vulnerability.