Author

Mitchell J. Geer

NYU Langone HealthORCID 0000-0003-1457-987XUnclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 2 Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:host-pathogen & immune response· 1 Organism:zebrafish· 1

Frequent Co-Authors

Benjamin G. NeeliD· 2 shared Amanda W. LundiD· 1 shared Cameron Genxuan LianiD· 1 shared Connor FosteriD· 1 shared Daniel Pushparaju Yeggoni· 1 shared Daniëlle T.J. WouterseniD· 1 shared Eli RothenbergiD· 1 shared Heidi StuhlmanniD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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2 shared papersCancer Biology

Benjamin G. NeelORCID

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2 papers

Recent·Most cited

Cancer BiologybioRxivPreprintJun 13, 2026

SHP2 binds directly to SOS1 to enable RAS activation

Toshiyuki Araki, Yashika Agrawal, Sachin S. Katti…·Wolfgang Peti

This paper identifies a PTP activity-independent mechanism by which SHP2 promotes RAS/ERK activation through direct binding to SOS1.

1 citation

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ImmunologyProceedings of the National Academy of SciencesPeer-reviewedJul 14, 2025

T Lymphocyte-Specific Deletion of SHP1 and SHP2 Promotes Activation-Induced Cell Death of CD4+ T Cells and Impairs Antitumor Response

Connor Foster, Jasper Du, Oscar J. Pundel…·Benjamin G. Neel

This study shows that deleting SHP1 and SHP2 in T cells increases CD4+ T cell activation-induced cell death and reduces antitumor responses, with SHP2 loss enhancing anti-PD-1 therapy.

10 citations

mouseexperimental assay

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