Jesse Jackson

The paper identifies a claustral neuron population that coordinates cortical and hippocampal activity during sleep to enhance memory consolidation.

This study maps opioid receptor distribution in the mouse claustrum-dorsal endopiriform using mFISH and single-cell analysis, revealing cell-type-specific patterns conserved in macaques.

This study shows that glucosylceramide-induced ectosomes transmit pathogenic α-synuclein between neurons in Parkinson's disease.

The paper shows ApoE2 and ApoE3Ch variants protect neurons by removing toxic peroxidated lipids, while ApoE4 worsens lipid-induced damage in rodent models.

This study shows that ACC inputs to the claustrum change from excitatory to inhibitory with age, linked to parvalbumin interneuron maturation in mice.

The paper introduces an immunolabeling method to reliably identify claustrum cells in mice across developmental stages using Nurr1/Nr2f2 and Tle4 markers.

This study investigates the role of claustrum projections to the anterior cingulate cortex in modulating acute and chronic inflammatory pain behaviors.

This study shows that modulating claustral activity in mice alters cortical dynamics, with inhibition increasing activity in anterior-medial regions and excitation decreasing it.

This study investigates how claustrum neurons interact with the cortex during different vigilance states, finding that their activity and communication efficacy are modulated by cortical synchronization and cholinergic tone.

This study investigates how social isolation affects microglia structure and neural activity in specific brain regions of male and female mice, revealing region- and sex-specific changes.

The paper identifies two excitatory neuron subtypes in the claustrum with distinct molecular and circuit properties, revealing a core-shell spatial arrangement that reconciles conflicting views on its extent.

The paper maps claustrocortical projection patterns in mice, revealing topographic organization and distinct functional modules.

This study investigates the role of postictal hypoperfusion/hypoxia in memory impairments after seizures, finding that synaptic plasticity is specifically affected while other network functions remain intact.

The paper shows that astrocytes detoxify fatty acids from hyperactive neurons through mitochondrial oxidation, protecting neurons from toxicity.