Author

Courtney W. Houchen

Oklahoma City VA Medical CenterORCID 0000-0003-2487-5242Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Cancer Biology· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Assay:electronic health records· 1 Research Focus:host-pathogen & immune response· 1 Research Focus:metabolism & bioenergetics· 1

Frequent Co-Authors

Albert Tran· 2 shared Chao JiangiD· 2 shared Constantin GeorgescuiD· 2 shared Jonathan D. WreniD· 2 shared Michael T KinteriD· 2 shared Phoebe Ohene-MarfoiD· 2 shared Sabira MohammediD· 2 shared Sathyaseelan S. DeepaiD· 2 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersCancer Biology

Albert Tran

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Cancer BiologyHepatologyPeer-reviewedAug 4, 2026

Hepatocyte MLKL Drives Obesity-Driven Hepatocellular Carcinoma Progression via Mitochondrial Dysfunction Independent of Necroptosis in MASLD

Phoebe Ohene-Marfo, Sabira Mohammed, Chao Jiang…·Sathyaseelan S. Deepa

This study shows that hepatocyte MLKL promotes obesity-driven MASLD-HCC through mitochondrial dysfunction and MFN2 suppression, independent of necroptosis.

1 citation

mousebulk RNA-seq

𝕏 PostBluesky

Cell BiologyAging CellPeer-reviewedJul 1, 2026

A Non-canonical Role for Hepatocyte MLKL in Promoting Mitochondrial Dysfunction and Senescence in the Aging Liver

Sabira Mohammed, Chao Jiang, Travis Pennington…·Sathyaseelan S. Deepa

This study shows that MLKL in hepatocytes promotes liver aging by inducing senescence and mitochondrial dysfunction without causing necroptosis.

3 citations

mouseproteomics

𝕏 PostBluesky