Author

Anja Kerksiek

University Hospital BonnORCID 0009-0002-6162-6106Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Signature Assays, Organisms & Methods

Click to filter · or open tag

Organism:mouse· 3 Assay:bulk RNA-seq· 2 Approach:experimental assay· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Approach:genetic perturbation / CRISPR· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

Dieter LütjohanniD· 2 shared Aaron Ernesto Marure-RojanoiD· 1 shared Alaa OthmaniD· 1 shared Aleksandar Arsović· 1 shared Anthony K. ShumiD· 1 shared Antoine RimbertiD· 1 shared Antonio Vázquez Cobá· 1 shared Arnold von EckardsteiniD· 1 shared

Activity by Year

2021-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 19 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersNeuroscience

Dieter LütjohannORCID

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Cell BiologyAtherosclerosisPeer-reviewedAug 25, 2026

The COPI coatomer influences LDL receptor activity, hepatic lipid storage, and apoB secretion

Grigorios Panteloglou, Jérôme Robert, Marieke Smit…·Arnold von Eckardstein

This study identifies COPI coatomer subunits that regulate LDL receptor activity, apoB secretion, and hepatic lipid storage, with genetic variants linked to elevated LDL-C levels.

DOI 10.1016/j.atherosclerosis.2026.121887

humanmouse

𝕏 PostBluesky

NeuroscienceEMBO Molecular MedicinePeer-reviewedAug 3, 2026

Reducing CETP activity prevents memory decline in an Alzheimer's disease mouse model

Jasmine Phénix, Isabel MacKenzie Sarty, Megan S. Katz…·Lisa Marie Munter

The paper shows that reducing CETP activity in Alzheimer's disease mice prevents memory decline and alters lipid and gene expression profiles, supporting CETP as a therapeutic target.

DOI 10.1038/s44321-026-00482-w

mouseproteomics

𝕏 PostBluesky

NeuroscienceNeurobiology of DiseasePeer-reviewedFeb 10, 2021

Atxn2-CAG100-KnockIn mouse spinal cord shows progressive TDP43 pathology associated with cholesterol biosynthesis suppression

Júlia Canet-Pons, Nesli-Ece Şen, Aleksandar Arsović…·Georg W. J. Auburger

This study examines an SCA2 mouse model to identify molecular mechanisms of spinal cord pathology, finding progressive TDP43 aggregation and cholesterol biosynthesis suppression.

54 citations

mousebulk RNA-seq

𝕏 PostBluesky