Author

A. L. H. Webster

Rockefeller University

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:cancer & tumor microenvironment· 2 Organism:human· 2 Assay:whole-genome sequencing· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:differential expression· 1

Frequent Co-Authors

S. I. Wells· 2 shared A. D. Auerbach· 1 shared A. G. Paulovich· 1 shared A. Goldfarb· 1 shared A. Koren· 1 shared A. Rosenberg· 1 shared A. Smogorzewska· 1 shared A.-M. Gonzalez· 1 shared

Activity by Year

2021-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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2 shared papersCancer Biology

S. I. Wells

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Author profile →

3 papers

Recent·Most cited

G-quadruplexes are a source of vulnerability in BRCA2 deficient granule cell progenitors and medulloblastoma

Danielle Keahi, Mathijs Arnoud Sanders, Matthew Robert Paul, A. L. H. Webster, Yin Fang, Tom F. Wiley, Samer Shalaby, Thomas S. Carroll, Settara C. Chandrasekharappa, Carolina Sandoval-Garcia, Margaret L. MacMillan, John E. Wagner, Mary E. Hatten, Agata Smogorzewska

This study identifies G-quadruplexes as a source of genome instability in BRCA2-deficient granule cell progenitors and medulloblastoma, suggesting PIF1 helicase as a therapeutic target.

Proceedings of the National Academy of Sciences · Aug 25, 2025Peer-reviewedCancer Biology

Fanconi anemia-isogenic head and neck cancer cell line pairs - a basic and translational science resource

H. T. Nguyen, W. Tang, A. L. H. Webster, J. R. Whiteaker, C. M. Chandler, R. Errazquin, L. B. Sullivan, E. Jonlin, E. E. Hoskins, E. Y. Chen, M. Fritzke, A. G. Paulovich, S. I. Wells, K. Roohollahi, J. Dorsman, , R. J. Monnat

The paper develops isogenic head and neck cancer cell line pairs to study Fanconi anemia-associated cancers and their treatment.

bioRxiv · Sep 11, 2022Cancer Biology

Fanconi Anemia Pathway Deficiency Drives Copy Number Variation in Squamous Cell Carcinomas

A. L. H. Webster, M. A. Sanders, K. Patel, R. Dietrich, R. J. Noonan, F. P. Lach, R. R. White, A. Goldfarb, K. Hadi, M. M. Edwards, F. X. Donovan, M. Jung, S. Sridhar, O. Fedrigo, H. Tian, , P. Campbell

This paper shows that FA pathway deficiency in squamous cell carcinomas leads to high structural variant rates and copy number alterations, linking genomic instability to tumor progression.

bioRxiv · Aug 14, 2021 · PreprintCancer Biology