Author

A. Q. Dinh

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:human· 2 Organism:bacteria (other)· 1 Research Focus:cell type annotation· 1 Approach:genetic perturbation / CRISPR· 1 Assay:proteomics· 1 Assay:whole-genome sequencing· 1

Frequent Co-Authors

C. A. Arias· 3 shared W. R. Miller· 3 shared A. Khan· 2 shared L. Diaz· 2 shared S. L. Egge· 2 shared T. T. Tran· 2 shared A. Nguyen· 1 shared A. Wanger· 1 shared

Activity by Year

2019-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 35 mutual co-author links

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3 shared papersMicrobiology

C. A. Arias

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Cefiderocol heteroresistance associated with mutations in TonB-dependent receptor genes in Pseudomonas aeruginosa of clinical origin

S. L. Egge, S. A. Rizvi, S. R. Simar, M. Alcade-Rico, J. R. Martinez, B. M. Hanson, A. Q. Dinh, R. de Paula Baptista, T. T. Tran, S. A. Shelburne, J. M. Munita, C. A. Arias, M. Hakki, W. R. Miller

This study examines TBDR mutations in P. aeruginosa clinical isolates and their association with cefiderocol resistance and heteroresistance.

bioRxiv · Jan 30, 2024 · PreprintMicrobiology

Membrane Lipids Augment Cell Envelope Stress Signaling and Resistance to Antibiotics and Antimicrobial Peptides in Enterococcus faecalis

W. R. Miller, A. Nguyen, K. V. Singh, S. Rizvi, A. Khan, S. G. Erickson, S. L. Egge, M. Cruz, A. Q. Dinh, L. Diaz, R. Zhang, L. Xu, D. A. Garsin, Y. Shamoo, C. A. Arias

This study identifies the MadRS system and its operon madEFG as mediators of resistance to antimicrobial peptides and antibiotics in Enterococcus faecalis, linking membrane lipid changes to stress response and virulence.

bioRxiv · Oct 17, 2023 · PreprintMicrobiology

Extremely Drug-Resistant Pseudomonas aeruginosa ST309 Harboring Tandem GES Enzymes: A Newly Emerging Threat in the United States

A. Khan, T. T. Tran, R. Rios, B. Hanson, W. C. Shropshire, Z. Sun, L. Diaz, A. Q. Dinh, A. Wanger, L. Ostrosky-Zeichner, T. Palzkill, C. A. Arias, W. R. Miller

The paper identifies two U.S. clinical isolates of Pseudomonas aeruginosa ST309 resistant to carbapenems and beta-lactam/beta-lactamase inhibitor combinations, harboring tandem GES 19 and 26 enzymes, and reports successful treatment with ceftazidime/avibactam plus aztreonam.

bioRxiv · Jan 30, 2019Microbiology