Author

A. Rosa Campos

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Assay:proteomics· 3 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1

Frequent Co-Authors

A. Bagchi· 1 shared A. C. McQuattie-Pimentel· 1 shared A. Deshpande· 1 shared A. V. Misharin· 1 shared B. Chen· 1 shared C. J. Eaves· 1 shared C.-C. Yang· 1 shared D. A. Wolf· 1 shared

Activity by Year

2019-2020 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 14 mutual co-author links

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1 shared paperCancer Biology

A. Bagchi

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

A JAK/STAT-Mediated Inflammatory Signaling Cascade Drives Oncogenesis In AF10-Rearranged AML

B. Chen, A. Deshpande, K. O. Barbosa, M. Kleppe, X. Lei, N. Yeddula, A. Rosa Campos, R. Wechsler-Reya, A. Bagchi, S. Meshinchi, C. J. Eaves, I. Jeremias, T. Haferlach, D. Frank, Z. Ronai, , A. Deshpande

This study identifies JAK1 as a therapeutic target in AF10-rearranged AML by showing its role in JAK/STAT-mediated inflammatory signaling and oncogenesis.

bioRxiv · Aug 31, 2020Cancer Biology

Proteostasis and Energetics as Proteome Hallmarks of Aging and Influenza Challenge in Pulmonary Disease

S. Loguercio, D. M. Hutt, A. Rosa Campos, T. Stoeger, R. A. Grant, A. C. McQuattie-Pimentel, H. Abdala-Valencia, Z. Lu, N. Joshi, K. Ridge, N. S. Chandel, J. I. Sznajder, R. I. Morimoto, A. V. Misharin, G. R. S. Budinger, W. E. Balch

This study examines proteome changes in alveolar macrophages and type 2 cells from young and old mice before and after influenza infection, revealing age-related proteostasis and mitochondrial dysfunction that worsen with infection.

bioRxiv · Sep 14, 2019 · PreprintCell Biology

Crosstalk between eIF2α and eEF2 phosphorylation pathways optimizes translational arrest in response to oxidative stress

M. Sanchez, Y. Lin, C.-C. Yang, P. McQuary, A. Rosa Campos, P. Aza Blanc, D. A. Wolf

This study shows that oxidative stress causes translational arrest through eEF2 phosphorylation, not eIF2, and reveals crosstalk between eIF2 and eEF2 pathways in mouse cells.

bioRxiv · Jul 5, 2019Cell Biology