Author

Abigail Sondrini

University of Massachusetts Chan Medical School

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Assay:proteomics· 2 Research Focus:cell signaling & trafficking· 1 Organism:human· 1 Organism:mouse· 1

Frequent Co-Authors

Kiera ClaytoniD· 2 shared Aaron ShulkiniD· 1 shared Alberto BosqueiD· 1 shared Alicja K. TrochaiD· 1 shared Alper KüçükuraliD· 1 shared Amanda Modica· 1 shared Fiachra HumphriesiD· 1 shared Komal Cheema· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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2 shared papersImmunology

Kiera ClaytonORCID

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2 papers

Recent·Most cited

Non-canonical TLR signaling restricts cytosolic LPS detection.

Shriram Ramani, Sara E. Cahill, Matthew Finnegan, Laurel Stine, Abigail Sondrini, Kiera Clayton, Liraz Shmuel-Galia, Fiachra Humphries

This study identifies MARCO as a decoy LPS sensor regulated by non-canonical TLR signaling, which restricts inflammasome activation and prevents excessive inflammation.

The Journal of Immunology · Jul 28, 2026Peer-reviewedImmunology

Loss of Vpr-driven TRAIL-R2 expression protects HIV-infected cells from non-canonical NK cell TRAIL attack

Paula E. Grasberger, Abigail Sondrini, Nicole C. Glidden, Amanda Modica, Natalie Pushlar, Seden Bedir, Tara Bromfield, Sheldon Gentling, Komal Cheema, Alper Küçükural, Milena Ozdemir, Maria L. Zapp, Alberto Bosque, Louise Leyre, Aaron Shulkin, , Kiera Clayton

The paper shows HIV uses Vpr to increase TRAIL-R2 expression in CD4+ T cells, which enhances NK cell killing through non-canonical TRAIL signaling.

bioRxiv · Apr 19, 2026 · PreprintImmunology