Author

C. Ribeyre

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 4 Cell Biology· 1 Plant Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 6 Organism:human· 5 Research Focus:cancer & tumor microenvironment· 3 Assay:bulk RNA-seq· 2 Research Focus:cell signaling & trafficking· 2 Assay:ChIP-seq· 2 Research Focus:chromatin & epigenetic regulation· 2 Assay:proteomics· 2

Frequent Co-Authors

A. Constantinou· 3 shared J.-S. Hoffmann· 2 shared M. Larroque· 2 shared M.-J. Pillaire· 2 shared R. Lebdy· 2 shared A. Aze· 1 shared A. El kaoutari· 1 shared A. Frances· 1 shared

Activity by Year

2019-2023 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 23 mutual co-author links

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3 shared papersCancer Biology

A. Constantinou

Mutual co-authors in cluster

Author profile →

6 papers

Recent·Most cited

Replication poison treated BRCA1-deficient breast cancers are prone to MRE11 over-resection resulting in single strand DNA accumulation and mitotic catastrophe

I. Tabet, E. Orhan, E. Candiello, L. Fenou, C. Velazquez, B. Orsetti, G. Rodier, W. Jacot, C. Ribeyre, C. Sardet, C. Theillet

This study shows BRCA1-deficient breast cancers are highly sensitive to gemcitabine, leading to ssDNA accumulation and mitotic catastrophe, which may enhance antitumor immune response.

bioRxiv · Oct 31, 2023 · PreprintCancer Biology

Cytidine deaminase protects pancreatic cancer cells from replicative stress and drives resistance to DNA-targeting drugs

A. Lumeau, N. Bery, A. Frances, M. Gayral, C. Ribeyre, C. Lopez, A. Nevot, A. El kaoutari, G. Labrousse, M. Madrid-Mencia, M.-J. Pillaire, V. Pancaldi, V. Bergoglio, N. J. V. Dusetti, J.-S. Hoffmann, , P. Cordelier

This study shows that Cytidine deaminase (CDA) protects pancreatic cancer cells from replication stress and contributes to resistance against DNA-targeting drugs.

bioRxiv · Oct 23, 2021 · PreprintCancer Biology

The organizer of chromatin topology RIF1 ensures cellular resilience to DNA replication stress.

C. Ribeyre, R. Lebdy, J. Patouillard, M. Larroque, R. Abou-Merhi, C. Larroque, A. Constantinou

This study shows RIF1 stabilizes chromatin topology to reduce DNA replication stress-induced genomic instability.

bioRxiv · Oct 22, 2021Cell Biology

KDM5 histone-demethylases contribute to replication stress response and tolerance.

M. Vandromme, S. Gaillard, V. Charasson, C. Ribeyre, K. Salifou, M.-J. Pillaire, J.-S. Hoffmann, A. Constantinou, D. Trouche

This study shows KDM5A and KDM5B contribute to replication stress response and tolerance through mechanisms independent of their demethylase activity.

bioRxiv · Dec 16, 2019 · PreprintCancer Biology

Experimental system and image analysis software for high throughput phenotyping of mycorrhizal growth response in Brachypodium distachyon

F. Maviane-Macia, C. Ribeyre, L. Buendia, M. Gaston, M. Khafif, F. Devoilles, N. Peeters, B. Lefebvre

The paper develops an experimental system and image analysis software to study mycorrhizal growth response in Brachypodium distachyon, finding that nitrogen and phosphorus together enhance positive growth response in some genotypes.

bioRxiv · Sep 23, 2019 · PreprintPlant Biology

Dihydropyrimidinase protects from DNA replication stress caused by cytotoxic metabolites.

J. Basbous, A. Aze, L. Chaloin, R. Lebdy, D. Hodroj, C. Ribeyre, M. Larroque, C. Shepard, B. Kim, A. Pruvost, J. Moreaux, D. Maiorano, M. Mechali, A. Constantinou

This study shows dihydropyrimidines are cytotoxic metabolites that cause DNA replication stress and DPCs, suggesting DHP inhibition could target solid tumors.

bioRxiv · Mar 5, 2019Cancer Biology