Author

C. Wilen

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 2 Immunology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 2 Organism:mouse· 2 Assay:proteomics· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Approach:deep learning· 1 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

J. WeiiD· 2 shared A. O. Kolawole· 1 shared B. Nelson· 1 shared C. Wobus· 1 shared C. Wu· 1 shared D. Klein· 1 shared E. A. Kennedy· 1 shared E. Hassan· 1 shared

Activity by Year

2019-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersImmunology

J. WeiORCID

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Author profile →

3 papers

Recent·Most cited

Monospecific and bispecific monoclonal SARS-CoV-2 neutralizing antibodies that maintain potency against B.1.617

L. Peng, Y. Hu, M. Mankowski, P. Ren, R. Chen, J. Wei, M. Zhao, T. Li, T. Tripler, L. Ye, R. Chow, Z. Fang, C. Wu, M. Dong, M. Cook, , S. Chen

The paper develops and characterizes monospecific and bispecific monoclonal antibodies that neutralize SARS-CoV-2 and its variants, including B.1.617.2 Delta.

bioRxiv · Dec 21, 2021Immunology

CD300lf is the primary physiologic receptor of murine norovirus but not human norovirus

V. Graziano, F. C. Walker, E. A. Kennedy, J. Wei, K. Ettayebi, M. Simoes, R. Filler, E. Hassan, L. L. Hsieh, A. O. Kolawole, C. Wobus, L. C. Lindesmith, R. S. Baric, M. Estes, R. C. Orchard, , C. Wilen

This paper identifies CD300lf as the primary receptor for murine norovirus in vivo but shows it is not used by human norovirus.

PLOS Pathogens · Apr 6, 2020Peer-reviewedMicrobiology

Mouse Norovirus infection arrests host cell translation uncoupled from the stress granule-PKR-eIF2α axis

S. Fritzlar, T. Aktepe, Y.-W. Chao, M. McAllaster, C. Wilen, P. White, J. Mackenzie

This study shows that mouse norovirus infection suppresses host translation and cytokine production while promoting viral replication by recruiting G3BP1 to block stress granule formation.

bioRxiv · Jan 31, 2019Microbiology