Author

David G. Schroeder

Jackson Laboratory

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 1 Genetics· 1

Signature Assays, Organisms & Methods

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Organism:mouse· 2 Research Focus:benchmarking· 1 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

Gregory A. CoxiD· 2 shared Abigail L. D. TadeneviD· 1 shared Brian Robert HoffmanniD· 1 shared Carlos Costas‐InsuaiD· 1 shared Carol J. BultiD· 1 shared Claudio A.P. JoazeiroiD· 1 shared Courtney L. HattoniD· 1 shared G C Murray· 1 shared

Activity by Year

2022-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersBiochemistry

Gregory A. CoxORCID

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Author profile →

2 papers

Recent·Most cited

Poly-alanine-tailing is a modifier of neurodegeneration caused by Listerin mutation

Hao‐Chih Hung, Carlos Costas‐Insua, Sarah E. Holbrook, Jennifer E. Stauffer, Paige B Martin, Tina Müller, David G. Schroeder, Yu Kigoshi-Tansho, Haifei Xu, Rüdiger Rudolf, Gregory A. Cox, Claudio A.P. Joazeiro

This study shows that impairing NEMF's poly-alanine-tailing activity in mice interacts with Listerin mutations to cause neurodegeneration, revealing a role for Ala-tailing in proteostasis and neurodegeneration.

bioRxiv · Aug 24, 2024 · PreprintBiochemistry

Mouse models of NADK2 deficiency analyzed for metabolic and gene expression changes to elucidate pathophysiology

G C Murray, Preeti Bais, Courtney L. Hatton, Abigail L. D. Tadenev, Brian Robert Hoffmann, Timothy J. Stodola, Kathryn H. Morelli, Samia L. Pratt, David G. Schroeder, R Doty, Oliver Fiehn, Simon W. M. John, Carol J. Bult, Gregory A. Cox, Robert W. Burgess

This study analyzes NADK2 deficiency in mouse models to identify metabolic and gene expression changes, revealing similarities and differences with another neurological disease model.

Human Molecular Genetics · Jul 7, 2022Peer-reviewedGenetics