Author

E. F. Petricoin

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

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Cancer Biology· 5

Signature Assays, Organisms & Methods

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Approach:experimental assay· 5 Organism:human· 4 Research Focus:cancer & tumor microenvironment· 3 Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Assay:proteomics· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell-cell communication· 1

Frequent Co-Authors

M. Pierobon· 3 shared A. Agarwal· 1 shared A. C. deCarvalho· 1 shared A. D. Cox· 1 shared A. J. Armstrong· 1 shared A. K. Sood· 1 shared A. L. Hunt· 1 shared A. Multani· 1 shared

Activity by Year

2017-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 13 mutual co-author links

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3 shared papersCancer Biology

M. Pierobon

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5 papers

Recent·Most cited

p53 modulates kinase inhibitor resistance and lineage plasticity in NF1-related MPNSTs

J. L. Grit, L. E. McGee, E. A. Tovar, C. J. Essenburg, E. Wolfrum, I. Beddows, K. Williams, R. Sheridan, J. Schipper, M. Adams, M. Arumugam, T. Vander Woude, S. Gurunathan, J. M. Field, J. Wulfkuhle, , M. Steensma

This study shows p53 loss in NF1-related MPNSTs leads to MET instability, kinome reprogramming, and therapy resistance through altered signaling and lineage plasticity.

bioRxiv · Jan 18, 2023Cancer Biology

Defining the KRAS-regulated kinome in KRAS-mutant pancreatic cancer

J. N. Diehl, J. E. Klomp, K. R. Snare, D. R. Blake, P. S. Hibshman, Z. D. Kaiser, T. S. K. Gilbert, E. Baldelli, M. Pierobon, B. Papke, R. Yang, R. G. Hodge, N. U. Rashid, E. F. Petricoin, L. E. Herring, , C. J. Der

This study identifies upregulated and downregulated kinases in KRAS-mutant pancreatic cancer using chemical proteomics, revealing potential therapeutic targets.

bioRxiv · Apr 27, 2021 · PreprintCancer Biology

Extensive Intratumor Proteogenomic Heterogeneity Revealed by Multiregion Sampling in High-Grade Serous Ovarian Tumor Specimens

A. L. Hunt, N. W. Bateman, W. Barakat, S. Makohon-Moore, B. L. Hood, K. A. Conrads, M. Zhou, V. Calvert, M. Pierobon, J. Loffredo, T. J. Litzi, J. Oliver, D. Mitchell, G. Gist, C. Rojas, , T. P. Conrads

The paper analyzes proteogenomic heterogeneity in ovarian tumors using multiregion sampling and reveals distinct expression patterns between tumor epithelium and stroma.

bioRxiv · Oct 6, 2020 · PreprintCancer Biology

Convergent evolution of p38/MAPK activation in hormone resistant prostate cancer mediates pro-survival, immune evasive, and metastatic phenotypes

K. E. Ware, S. Gupta, J. Eng, G. Kemeny, B. J. Puviindran, W.-C. Foo, L. A. Crawford, R. G. Almquist, D. Runyambo, B. C. Thomas, M. U. Sheth, A. Agarwal, M. Pierobon, E. F. Petricoin, D. L. Corcoran, , J. Somarelli

This study identifies p38/MAPK pathway activation as a convergent mechanism driving survival, immune evasion, and metastasis in hormone-resistant prostate cancer.

bioRxiv · Apr 22, 2020 · PreprintCancer Biology

Discordant inheritance of chromosomal and extrachromosomal DNA elements contributes to dynamic disease evolution in glioblastoma

A. C. deCarvalho, H. Kim, L. M. Poisson, M. E. Winn, C. Mueller, D. Cherba, J. Koeman, S. Seth, A. Protopopov, M. Felicella, S. Zheng, A. Multani, Y. Zhang, J. Zhang, D.-H. Nam, , R. Verhaak

This study shows that extrachromosomal DNA elements and chromosomal alterations in glioblastoma have distinct inheritance patterns and contribute to genomic heterogeneity during tumor evolution.

bioRxiv · Jun 27, 2017Cancer Biology